Beate Rohde-Schulz

11 papers receiving 333 citations

Peers

Beate Rohde-Schulz
Comparison fields: 5 of 63
  • Genetics 181
  • Immunology and Allergy 30
  • Reproductive Medicine 36
  • Endocrinology, Diabetes and Metabolism 68
  • Oncology 64
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Maï Thu Vu Hai France
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Citations per year

Countries citing papers authored by Beate Rohde-Schulz

Since Specialization
Citations

This map shows the geographic impact of Beate Rohde-Schulz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beate Rohde-Schulz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beate Rohde-Schulz more than expected).

Fields of papers citing papers by Beate Rohde-Schulz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Beate Rohde-Schulz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beate Rohde-Schulz. The network helps show where Beate Rohde-Schulz may publish in the future.

Co-authors

The 25 scholars most cited alongside Beate Rohde-Schulz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Beate Rohde-Schulz Line = papers co-authored together Beate Rohde-Schulz links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 2008216
2
Ligand mediated activation of ectopic EGF receptor promotes matrix protein adhesion and lung colonization of rat mammary adenocarcinoma cells.
199548
3 199416
4 201513
5 19909
6 19929
7
EGF enhances attachment of metastatic rat mammary adenocarcinoma cell clone MTLn3 to fibronectin and collagen.
19959
8 19855
9 19875
10
Exacerbation of human immunodeficiency virus infection in promonocytic cells by bacterial immunomodulators.
19905
11 20083

About Beate Rohde-Schulz

Beate Rohde-Schulz is a scholar working on Molecular Biology, Oncology, Epidemiology, Endocrinology, Diabetes and Metabolism and Genetics, having authored 11 papers that have together received 338 indexed citations. Recurring topics across this work include Antimicrobial Peptides and Activities (3 papers), HER2/EGFR in Cancer Research (3 papers), Estrogen and related hormone effects (2 papers), Radiopharmaceutical Chemistry and Applications (2 papers), Reproductive System and Pregnancy (2 papers), Influenza Virus Research Studies (2 papers), Pneumocystis jirovecii pneumonia detection and treatment (1 paper) and Viral Infectious Diseases and Gene Expression in Insects (1 paper). The work is most often cited by research in Genetics (181 citations), Immunology and Allergy (30 citations), Reproductive Medicine (36 citations), Endocrinology, Diabetes and Metabolism (68 citations) and Oncology (64 citations). Beate Rohde-Schulz has collaborated with scholars based in Germany and United States. Frequent co-authors include Iris Fuchs, Christiane Otto, Gernot Langer, Karl‐Heinrich Fritzemeier, Dominic E. A. Brittain, Katja Prelle, Reinhard Nubbemeyer, Benjamin Bader, K. Noel Masihi and Andreas M. Kaufmann. Their work appears in journals such as Endocrinology, Advances in experimental medicine and biology, Cancer Immunology Immunotherapy, AIDS Research and Human Retroviruses and International Journal of Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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