Barbara Bodner
Impact in
- Oncology top 10%
- Cancer Immunotherapy and Biomarkers
- Cancer-related Molecular Pathways
- Immunology top 10%
- Immunotherapy and Immune Responses
Papers in
-
- Cell death mechanisms and regulation 3
- Ubiquitin and proteasome pathways 1
- Oncology 3
- Cancer-related Molecular Pathways 2
- Co-authors
- Brian J. Nickoloff (6 shared papers)Jian‐Zhong Qin (3 shared papers)Vijaya Chaturvedi (4 shared papers)Mary J.C. Hendrix (2 shared papers)Lucio Miele (2 shared papers)Jeffrey M. Trent (2 shared papers)Frank Bennett (2 shared papers)Lawrence Stennett (2 shared papers)
- Journals
- Cancer Research (2 papers)Molecular Cancer Therapeutics (1 paper)Journal of Dermatological Science (1 paper)Gastroenterology (1 paper)Oncogene (1 paper)
- Partner nations
- United States
In The Last Decade
Barbara Bodner
9 papers receiving 844 citations
Peers
Comparison fields: 5 of 75
- Oncology 313
- Immunology 232
- Hematology 75
- Molecular Biology 421
- Cancer Research 82
Countries citing papers authored by Barbara Bodner
This map shows the geographic impact of Barbara Bodner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Barbara Bodner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Barbara Bodner more than expected).
Fields of papers citing papers by Barbara Bodner
This network shows the impact of papers produced by Barbara Bodner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Barbara Bodner. The network helps show where Barbara Bodner may publish in the future.
Co-authors
The 25 scholars most cited alongside Barbara Bodner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2001 | 328 | |
| 2 | 2005 | 271 | |
| 3 | 2004 | 108 | |
| 4 | 2004 | 51 | |
| 5 | 2005 | 41 | |
| 6 | 2006 | 28 | |
| 7 | 2005 | 19 | |
| 8 | 2005 | 11 | |
| 9 | 2012 | 3 |
About Barbara Bodner
Barbara Bodner is a scholar working on Molecular Biology, Oncology, Pathology and Forensic Medicine, Radiology, Nuclear Medicine and Imaging and Infectious Diseases, having authored 9 papers that have together received 860 indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (3 papers), Corneal Surgery and Treatments (2 papers), Cancer-related Molecular Pathways (2 papers), Cancer Mechanisms and Therapy (1 paper), Biomedical Research and Pathophysiology (1 paper), Prostate Cancer Treatment and Research (1 paper), Ubiquitin and proteasome pathways (1 paper) and Telomeres, Telomerase, and Senescence (1 paper). The work is most often cited by research in Oncology (313 citations), Immunology (232 citations), Hematology (75 citations), Molecular Biology (421 citations) and Cancer Research (82 citations). Barbara Bodner has collaborated with scholars based in United States. Frequent co-authors include Brian J. Nickoloff, Jian‐Zhong Qin, Vijaya Chaturvedi, Mary J.C. Hendrix, Lucio Miele, Jeffrey M. Trent, Frank Bennett, Lawrence Stennett, Pamela M. Pollock and Eva M. Wojcik. Their work appears in journals such as Cancer Research, Molecular Cancer Therapeutics, Journal of Dermatological Science, Gastroenterology and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.