Ariane Watson
Impact in
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- Epigenetics and DNA Methylation
- Genomics and Chromatin Dynamics
- Cancer-related gene regulation
- RNA modifications and cancer
- CRISPR and Genetic Engineering
- Pluripotent Stem Cells Research
- Histone Deacetylase Inhibitors Research
Papers in
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- Genomics and Chromatin Dynamics 4
- Epigenetics and DNA Methylation 4
- Cancer-related gene regulation 2
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- Nerve injury and regeneration 1
- Co-authors
- Gerard Cagney (6 shared papers)Giorgio Oliviero (5 shared papers)Adrian P. Bracken (3 shared papers)Gundula Streubel (3 shared papers)Kieran Wynne (3 shared papers)Andrea Scelfo (2 shared papers)Darren J. Fitzpatrick (2 shared papers)Eugène Dillon (2 shared papers)
- Journals
- Molecular Cell (2 papers)Scientific Reports (2 papers)Developmental Biology (1 paper)The FASEB Journal (1 paper)The EMBO Journal (1 paper)
- Partner nations
- IrelandUnited StatesItaly
In The Last Decade
Ariane Watson
7 papers receiving 249 citations
Peers
Comparison fields: 5 of 50
- Molecular Biology 211
- Developmental Neuroscience 7
- Aging 3
- Virology 5
- Genetics 28
Countries citing papers authored by Ariane Watson
This map shows the geographic impact of Ariane Watson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ariane Watson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ariane Watson more than expected).
Fields of papers citing papers by Ariane Watson
This network shows the impact of papers produced by Ariane Watson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ariane Watson. The network helps show where Ariane Watson may publish in the future.
Co-authors
The 25 scholars most cited alongside Ariane Watson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 125 | |
| 2 | 2017 | 39 | |
| 3 | 2015 | 36 | |
| 4 | 2016 | 20 | |
| 5 | 2020 | 16 | |
| 6 | 2019 | 10 | |
| 7 | 2017 | 3 |
About Ariane Watson
Ariane Watson is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Virology, Pulmonary and Respiratory Medicine and Genetics, having authored 7 papers that have together received 249 indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (4 papers), Epigenetics and DNA Methylation (4 papers), Cancer-related gene regulation (2 papers), Genetics and Neurodevelopmental Disorders (1 paper), MicroRNA in disease regulation (1 paper), HIV Research and Treatment (1 paper), Nerve injury and regeneration (1 paper) and interferon and immune responses (1 paper). The work is most often cited by research in Molecular Biology (211 citations), Developmental Neuroscience (7 citations), Aging (3 citations), Virology (5 citations) and Genetics (28 citations). Ariane Watson has collaborated with scholars based in Ireland, United States and Italy. Frequent co-authors include Gerard Cagney, Giorgio Oliviero, Adrian P. Bracken, Gundula Streubel, Kieran Wynne, Andrea Scelfo, Darren J. Fitzpatrick, Eugène Dillon, Diego Pasini and Gian Luca Negri. Their work appears in journals such as Molecular Cell, Scientific Reports, Developmental Biology, The FASEB Journal and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.