Anders Bratt
Impact in
- Cell Biology top 5%
- Hippo pathway signaling and YAP/TAZ
- Cellular Mechanics and Interactions
- Cancer Research top 10%
- MicroRNA in disease regulation
- Cancer Genomics and Diagnostics
Papers in
-
- Hippo pathway signaling and YAP/TAZ 5
-
- Angiogenesis and VEGF in Cancer 3
- Cancer-related gene regulation 1
- Cell death mechanisms and regulation 1
- Co-authors
- Lars Holmgren (6 shared papers)M. Judah Folkman (1 shared paper)Anna‐Lena Spetz (1 shared paper)Anna Szeles (1 shared paper)Marie Arsenian‐Henriksson (1 shared paper)Anna Bergsmedh (1 shared paper)Karin Aase (4 shared papers)Niina Veitonmäki (2 shared papers)
- Journals
- FEBS Journal (1 paper)Gene (1 paper)Proceedings of the National Academy of Sciences (1 paper)Journal of Cell Science (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- SwedenUnited States
In The Last Decade
Anders Bratt
6 papers receiving 830 citations
Peers
Comparison fields: 5 of 74
- Cell Biology 279
- Cancer Research 236
- Molecular Biology 575
- Immunology and Allergy 46
- Immunology 141
Countries citing papers authored by Anders Bratt
This map shows the geographic impact of Anders Bratt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anders Bratt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anders Bratt more than expected).
Fields of papers citing papers by Anders Bratt
This network shows the impact of papers produced by Anders Bratt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anders Bratt. The network helps show where Anders Bratt may publish in the future.
Co-authors
The 16 scholars most cited alongside Anders Bratt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2001 | 432 | |
| 2 | 2005 | 129 | |
| 3 | 2002 | 117 | |
| 4 | 2006 | 95 | |
| 5 | 2004 | 39 | |
| 6 | 2003 | 37 |
About Anders Bratt
Anders Bratt is a scholar working on Cell Biology, Molecular Biology, Immunology and Allergy, Neurology and Biotechnology, having authored 6 papers that have together received 849 indexed citations. Recurring topics across this work include Hippo pathway signaling and YAP/TAZ (5 papers), Angiogenesis and VEGF in Cancer (3 papers), Cell Adhesion Molecules Research (2 papers), Vascular Malformations Diagnosis and Treatment (1 paper), Cancer-related gene regulation (1 paper), Phagocytosis and Immune Regulation (1 paper), Cell death mechanisms and regulation (1 paper) and Cancer Research and Treatments (1 paper). The work is most often cited by research in Cell Biology (279 citations), Cancer Research (236 citations), Molecular Biology (575 citations), Immunology and Allergy (46 citations) and Immunology (141 citations). Anders Bratt has collaborated with scholars based in Sweden and United States. Frequent co-authors include Lars Holmgren, M. Judah Folkman, Anna‐Lena Spetz, Anna Szeles, Marie Arsenian‐Henriksson, Anna Bergsmedh, Karin Aase, Niina Veitonmäki, Boris Troyanovsky and Olivier Birot. Their work appears in journals such as FEBS Journal, Gene, Proceedings of the National Academy of Sciences, Journal of Cell Science and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.