Amparo Cano

31.7k citations
134 papers · 22.2k · 10 hit papers · h-index 59

Impact in

  • Oncology top 0.05%
    • Cancer Cells and Metastasis
    • Cancer-related molecular mechanisms research
    • MicroRNA in disease regulation
    • Cancer, Hypoxia, and Metabolism

Papers in

    • Wnt/β-catenin signaling in development and cancer 47
    • Cancer-related gene regulation 27
    • Kruppel-like factors research 17
    • Microbial metabolism and enzyme function 17
    • Epigenetics and DNA Methylation 14
    • TGF-β signaling in diseases 7
    • Cancer Cells and Metastasis 38

Amparo Cano

134 papers receiving 21.8k citations

Amparo Cano's Hit Papers

Metastatic Colonization Requires the Repression of the Epithelial-Mesenchymal Transition Inducer Prrx1 2012 · 762 citations
7620+15+30Years since publication50010001.5k2.0k2.5k

Peers

Amparo Cano
Comparison fields: 5 of 148
  • Oncology 8.7k
  • Cancer Research 4.2k
  • Molecular Biology 14.5k
  • Cell Biology 2.3k
  • Immunology and Allergy 644
Replace Thomas Brabletz with:
Thomas Brabletz Germany
Allan Balmain United States
Gerhard Christofori Switzerland
Hong Wu United States
Owen J. Sansom United Kingdom
Eduard Batlle Spain
Mary W. Brooks United States
Geert Berx Belgium
Ruggero De Maria Italy
Amparo Cano relative to Thomas Brabletz Germany Thomas Brabletz's profile →
Citations per field
00.5×1.5×
Thomas Brabletz · 1×
Citations per year

Countries citing papers authored by Amparo Cano

Since Specialization
Citations

This map shows the geographic impact of Amparo Cano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amparo Cano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amparo Cano more than expected).

Fields of papers citing papers by Amparo Cano

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Amparo Cano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amparo Cano. The network helps show where Amparo Cano may publish in the future.

Co-authors

The 25 scholars most cited alongside Amparo Cano, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Amparo Cano Line = papers co-authored together Amparo Cano links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 134 papers — load more, or switch the sort, to bring in the rest.

#Work
1
The transcription factor Snail controls epithelial–mesenchymal transitions by repressing E-cadherin expression
Hit paper breakdown →
20002985
2
Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype?
Hit paper breakdown →
20072619
3
DNA-dependent transcription of adenovirus genes in a soluble whole-cell extract.
Hit paper breakdown →
19801149
4
The transcription factor Slug repressesE-cadherinexpression and induces epithelial to mesenchymal transitions: a comparison with Snail and E47 repressors
Hit paper breakdown →
2002933
5
Epithelial-Mesenchymal Transition in Breast Cancer Relates to the Basal-like Phenotype
Hit paper breakdown →
2008845
6
Metastatic Colonization Requires the Repression of the Epithelial-Mesenchymal Transition Inducer Prrx1
Hit paper breakdown →
2012762
7
DeltaEF1 is a transcriptional repressor of E-cadherin and regulates epithelial plasticity in breast cancer cells
Hit paper breakdown →
2005658
8
Vitamin D3 promotes the differentiation of colon carcinoma cells by the induction of E-cadherin and the inhibition of β-catenin signaling
Hit paper breakdown →
2001646
9
Snail Mediates E-Cadherin Repression by the Recruitment of the Sin3A/Histone Deacetylase 1 (HDAC1)/HDAC2 Complex
Hit paper breakdown →
2003607
10
Transforming Growth Factor β-1 Induces Snail Transcription Factor in Epithelial Cell Lines
Hit paper breakdown →
2003552
11 2008488
12 2008482
13 2002481
14 2004467
15 2005372
16 2001362
17
Correlation of E-cadherin expression with differentiation grade and histological type in breast carcinoma.
1993346
18 2008291
19 2006262
20 2006242

About Amparo Cano

Amparo Cano is a scholar working on Molecular Biology, Oncology, Cell Biology, Cancer Research and Genetics, having authored 134 papers that have together received 22.2k indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (47 papers), Cancer Cells and Metastasis (38 papers), Cancer-related gene regulation (27 papers), Kruppel-like factors research (17 papers), Microbial metabolism and enzyme function (17 papers), Epigenetics and DNA Methylation (14 papers), Skin and Cellular Biology Research (9 papers) and TGF-β signaling in diseases (7 papers). The work is most often cited by research in Oncology (8.7k citations), Cancer Research (4.2k citations), Molecular Biology (14.5k citations), Cell Biology (2.3k citations) and Immunology and Allergy (644 citations). Amparo Cano has collaborated with scholars based in Spain, United States and France. Frequent co-authors include Héctor Peinado, Francisco Portillo, David Olmeda, Gema Moreno‐Bueno, M. Ángela Nieto, Mirna Pérez‐Moreno, Annamaria Locascio, Isabel Rodrigo, Marı́a José Blanco and Miguel Quintanilla. Their work appears in journals such as Cancer Research, Oncogene, Journal of Cell Science, Experimental Cell Research and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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