Amina Rafiki
Impact in
- Developmental Neuroscience top 5%
- Neurogenesis and neuroplasticity mechanisms
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- Neuroscience and Neuropharmacology Research
Papers in
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- Neuroscience and Neuropharmacology Research 10
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- Ion channel regulation and function 5
- Receptor Mechanisms and Signaling 2
- Ion Transport and Channel Regulation 1
- Co-authors
- Linda H. Bergersen (3 shared papers)Ole Petter Ottersen (2 shared papers)Jean‐Luc Boulland (3 shared papers)Andrew P. Halestrap (1 shared paper)Farrukh A. Chaudhry (2 shared papers)Jon Storm‐Mathisen (2 shared papers)Alfonso Represa (3 shared papers)Vidar Gundersen (1 shared paper)
In The Last Decade
Amina Rafiki
10 papers receiving 806 citations
Peers
Comparison fields: 5 of 70
- Developmental Neuroscience 119
- Cellular and Molecular Neuroscience 532
- Neurology 92
- Biochemistry 76
- Clinical Biochemistry 54
Countries citing papers authored by Amina Rafiki
This map shows the geographic impact of Amina Rafiki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Amina Rafiki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Amina Rafiki more than expected).
Fields of papers citing papers by Amina Rafiki
This network shows the impact of papers produced by Amina Rafiki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Amina Rafiki. The network helps show where Amina Rafiki may publish in the future.
Co-authors
The 19 scholars most cited alongside Amina Rafiki, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2004 | 241 | |
| 2 | 2003 | 193 | |
| 3 | 2002 | 111 | |
| 4 | 1999 | 73 | |
| 5 | 1998 | 69 | |
| 6 | 2003 | 59 | |
| 7 | 1998 | 29 | |
| 8 | 1998 | 24 | |
| 9 | 2000 | 14 | |
| 10 | 1997 | 11 |
About Amina Rafiki
Amina Rafiki is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Psychiatry and Mental health, Pediatrics, Perinatology and Child Health and Cognitive Neuroscience, having authored 10 papers that have together received 824 indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (10 papers), Ion channel regulation and function (5 papers), Epilepsy research and treatment (2 papers), Receptor Mechanisms and Signaling (2 papers), Genetics and Neurodevelopmental Disorders (1 paper), Neurogenesis and neuroplasticity mechanisms (1 paper), Ion Transport and Channel Regulation (1 paper) and Metabolism and Genetic Disorders (1 paper). The work is most often cited by research in Developmental Neuroscience (119 citations), Cellular and Molecular Neuroscience (532 citations), Neurology (92 citations), Biochemistry (76 citations) and Clinical Biochemistry (54 citations). Amina Rafiki has collaborated with scholars based in France, Norway and Belgium. Frequent co-authors include Linda H. Bergersen, Ole Petter Ottersen, Jean‐Luc Boulland, Andrew P. Halestrap, Farrukh A. Chaudhry, Jon Storm‐Mathisen, Alfonso Represa, Vidar Gundersen, Robert H. Edwards and Rebecca P. Seal. Their work appears in journals such as The Journal of Comparative Neurology, European Journal of Neuroscience, Neuroscience, Journal of Neurochemistry and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.