Alex Osmand
Impact in
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- Genetic Neurodegenerative Diseases
- Neurology top 5%
- Neurological disorders and treatments
Papers in
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- Genetic Neurodegenerative Diseases 6
- Hereditary Neurological Disorders 1
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- Muscle Physiology and Disorders 4
- Mitochondrial Function and Pathology 3
- Co-authors
- Steven Finkbeiner (3 shared papers)Xiaofeng Gu (3 shared papers)X. William Yang (3 shared papers)Erin R. Greiner (2 shared papers)Joan S. Steffan (2 shared papers)Leslie M. Thompson (1 shared paper)Ronald Wetzel (1 shared paper)Rakesh K. Mishra (1 shared paper)
- Journals
- Neuron (2 papers)Journal of Clinical Investigation (1 paper)Journal of Huntington s Disease (1 paper)Journal of Biological Chemistry (1 paper)Human Molecular Genetics (1 paper)
- Partner nations
- United StatesUnited KingdomFrance
In The Last Decade
Alex Osmand
7 papers receiving 789 citations
Peers
Comparison fields: 5 of 46
- Cellular and Molecular Neuroscience 682
- Neurology 203
- Molecular Biology 625
- Aging 8
- Cell Biology 69
Countries citing papers authored by Alex Osmand
This map shows the geographic impact of Alex Osmand's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alex Osmand with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alex Osmand more than expected).
Fields of papers citing papers by Alex Osmand
This network shows the impact of papers produced by Alex Osmand. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alex Osmand. The network helps show where Alex Osmand may publish in the future.
Co-authors
The 25 scholars most cited alongside Alex Osmand, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2010 | 259 | |
| 2 | 2009 | 245 | |
| 3 | 2011 | 109 | |
| 4 | 2010 | 100 | |
| 5 | 2013 | 43 | |
| 6 | 2016 | 37 | |
| 7 | 1994 | 4 |
About Alex Osmand
Alex Osmand is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Neurology, Pulmonary and Respiratory Medicine and Epidemiology, having authored 7 papers that have together received 797 indexed citations. Recurring topics across this work include Genetic Neurodegenerative Diseases (6 papers), Muscle Physiology and Disorders (4 papers), Mitochondrial Function and Pathology (3 papers), Cerebrovascular and Carotid Artery Diseases (1 paper), Parkinson's Disease Mechanisms and Treatments (1 paper), Transcranial Magnetic Stimulation Studies (1 paper), Hereditary Neurological Disorders (1 paper) and Autophagy in Disease and Therapy (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (682 citations), Neurology (203 citations), Molecular Biology (625 citations), Aging (8 citations) and Cell Biology (69 citations). Alex Osmand has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include Steven Finkbeiner, Xiaofeng Gu, X. William Yang, Erin R. Greiner, Joan S. Steffan, Leslie M. Thompson, Ronald Wetzel, Rakesh K. Mishra, Ravindra Kodali and Andreas Weiss. Their work appears in journals such as Neuron, Journal of Clinical Investigation, Journal of Huntington s Disease, Journal of Biological Chemistry and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.