Alberto Bertollini

36 papers receiving 714 citations

Peers

Alberto Bertollini
Comparison fields: 5 of 84
  • Cellular and Molecular Neuroscience 398
  • Cell Biology 170
  • Biological Psychiatry 16
  • Molecular Biology 407
  • Physiology 101
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Countries citing papers authored by Alberto Bertollini

Since Specialization
Citations

This map shows the geographic impact of Alberto Bertollini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Alberto Bertollini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Alberto Bertollini more than expected).

Fields of papers citing papers by Alberto Bertollini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Alberto Bertollini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Alberto Bertollini. The network helps show where Alberto Bertollini may publish in the future.

Co-authors

The 25 scholars most cited alongside Alberto Bertollini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Alberto Bertollini Line = papers co-authored together Alberto Bertollini links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1977171
2 1975127
3 197649
4 197747
5
Regional differences in the synaptosomal uptake of 3H-gamma-aminobutyric acid and 14C-glutamate and possible role of exchange processes.
197446
6 198536
7 197626
8 198026
9 198921
10 199917
11 198817
12 199317
13 199316
14 197216
15 198514
16 198510
17
Stabilization of the T-state of ferrous human adult haemoglobin by chlorpromazine and trifluoperazine.
199910
18 19749
19 19887
20 19996

About Alberto Bertollini

Alberto Bertollini is a scholar working on Cell Biology, Molecular Biology, Cellular and Molecular Neuroscience, Physiology and Pediatrics, Perinatology and Child Health, having authored 36 papers that have together received 751 indexed citations. Recurring topics across this work include Hemoglobin structure and function (17 papers), Neuroscience and Neuropharmacology Research (6 papers), Neonatal Health and Biochemistry (6 papers), Erythrocyte Function and Pathophysiology (6 papers), Electron Spin Resonance Studies (5 papers), Neurotransmitter Receptor Influence on Behavior (4 papers), Receptor Mechanisms and Signaling (3 papers) and Protease and Inhibitor Mechanisms (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (398 citations), Cell Biology (170 citations), Biological Psychiatry (16 citations), Molecular Biology (407 citations) and Physiology (101 citations). Alberto Bertollini has collaborated with scholars based in Italy, France and United Kingdom. Frequent co-authors include Maurizio Raiteri, Giulio Levi, Renata Del Carmine, Francesco Angelini, Gino Amiconi, Maurizio Brunori, Paolo Ascenzi, Massimo Coletta, John Chen and Roberto Santucci. Their work appears in journals such as Journal of Inorganic Biochemistry, Journal of Biological Chemistry, Advances in experimental medicine and biology, Journal of Pharmacy and Pharmacology and European Journal of Biochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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