Xiaolan Du

2.5k citations

51 papers · 1.5k · h-index 21

Impact in

Rheumatology top 2%
- Osteoarthritis Treatment and Mechanisms
Cancer Research top 10%
- MicroRNA in disease regulation

Papers in

Molecular Biology 30
- Fibroblast Growth Factor Research 20
- Bone Metabolism and Diseases 4
- TGF-β signaling in diseases 3
Genetics 14
- Connective tissue disorders research 11
- Cleft Lip and Palate Research 3
- Craniofacial Disorders and Treatments 3

Co-authors: Lin Chen (34 shared papers)Yangli Xie (20 shared papers)Hangang Chen (14 shared papers)Nan Su (22 shared papers)Siru Zhou (12 shared papers)Zhenhong Ni (9 shared papers)Junjie Ouyang (4 shared papers)Qiaoyan Tan (14 shared papers)
Journals: International Journal of Biological Sciences (7 papers)Physical review. B. (2 papers)Autophagy (2 papers)Biochemical and Biophysical Research Communications (2 papers)Journal of Bone and Mineral Research (2 papers)
Partner nations: China United States Australia

In The Last Decade

Xiaolan Du

48 papers receiving 1.5k citations

Peers

Countries citing papers authored by Xiaolan Du

Since Specialization

Citations

This map shows the geographic impact of Xiaolan Du's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Xiaolan Du with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Xiaolan Du more than expected).

Fields of papers citing papers by Xiaolan Du

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Xiaolan Du. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Xiaolan Du. The network helps show where Xiaolan Du may publish in the future.

Co-authors

The 25 scholars most cited alongside Xiaolan Du, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Xiaolan Du Line = papers co-authored together Xiaolan Du links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 51 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Exosomes: roles and therapeutic potential in osteoarthritis Bone Research ·Zhenhong Ni, Siru Zhou, Song Li, Liang Kuang, Hangang Chen, Xiaoqing Luo, Junjie Ouyang, Mei He, Xiaolan Du, Lin Chen	2020	224
2	The exosome-like vesicles from osteoarthritic chondrocyte enhanced mature IL-1β production of macrophages and aggravated synovitis in osteoarthritis Cell Death and Disease ·Zhenhong Ni, Liang Kuang, Hangang Chen, Yangli Xie, Bin Zhang, Junjie Ouyang, Jiangyi Wu, Siru Zhou, Liang Chen, Nan Su, Qiaoyan Tan, Xiaoqing Luo, Bo Chen, Daiwen Chen, Liangjun Yin, Haiyang Huang, Xiaolan Du, Lin Chen	2019	145
3	Role of FGFs/FGFRs in skeletal development and bone regeneration Journal of Cellular Physiology ·Xiaolan Du, Yangli Xie, Cory J. Xian, Lin Chen	2012	132
4	A Pro253Arg mutation in fibroblast growth factor receptor 2 (Fgfr2) causes skeleton malformation mimicking human Apert syndrome by affecting both chondrogenesis and osteogenesis Bone ·Liangjun Yin, Xiaolan Du, Cuiling Li, Xiaoling Xu, Zhi Chen, Nan Su, Ling Zhao, Huabing Qi, Fubing Li, Jing Xue, Jing Yang, Min Jin, (unknown), Lin Chen	2007	115
5	SQSTM1-dependent autophagic degradation of PKM2 inhibits the production of mature IL1B/IL-1β and contributes to LIPUS-mediated anti-inflammatory effect Autophagy ·Bin Zhang, Hangang Chen, Junjie Ouyang, Yangli Xie, Liang Chen, Qiaoyan Tan, Xiaolan Du, Nan Su, Zhenhong Ni, Lin Chen	2019	98
6	Genetic inhibition of fibroblast growth factor receptor 1 in knee cartilage attenuates the degeneration of articular cartilage in adult mice Arthritis & Rheumatism ·Tujun Weng, Lingxian Yi, Junlan Huang, Fengtao Luo, Xuan Wen, Xiaolan Du, Qian Chen, (unknown), Di Chen, Lin Chen	2012	71
7	Fibroblast Growth Factor Receptor 3 Inhibits Osteoarthritis Progression in the Knee Joints of Adult Mice Arthritis & Rheumatology ·Junzhou Tang, Nan Su, Siru Zhou, Yangli Xie, Junlan Huang, Xuan Wen, Zuqiang Wang, Quan Wang, Wei Xu, Xiaolan Du, Hangang Chen, Lin Chen	2016	58
8	FGFR3/fibroblast growth factor receptor 3 inhibits autophagy through decreasing the ATG12–ATG5 conjugate, leading to the delay of cartilage development in achondroplasia Autophagy ·Xiaofeng Wang, Huabing Qi, Quan Wang, Ying Zhu, Xianxing Wang, Min Jin, Qiaoyan Tan, Qizhao Huang, Wei Xu, Xiaogang Li, Liang Kuang, Yubing Tang, Xiaolan Du, Di Chen, Lin Chen	2015	53
9	Chondrocyte FGFR3 Regulates Bone Mass by Inhibiting Osteogenesis Journal of Biological Chemistry ·Xuan Wen, Xiaogang Li, Yubin Tang, Junzhou Tang, Siru Zhou, Yangli Xie, Jingyuan Guo, Jing Yang, Xiaolan Du, Nan Su, Lin Chen	2016	47
10	FGFR3 Deficiency Causes Multiple Chondroma-like Lesions by Upregulating Hedgehog Signaling PLoS Genetics ·Siru Zhou, Yangli Xie, Junzhou Tang, Junlan Huang, Qizhao Huang, Wei Xu, Zuqiang Wang, Fengtao Luo, Quan Wang, Hangang Chen, Xiaolan Du, Yue Shen, Di Chen, Lin Chen	2015	42
11	Conditional Deletion of Fgfr3 in Chondrocytes leads to Osteoarthritis-like Defects in Temporomandibular Joint of Adult Mice Scientific Reports ·Siru Zhou, Yangli Xie, Wei Li, Junlan Huang, Zuqiang Wang, Junzhou Tang, Wei Xu, Xianding Sun, Qiaoyan Tan, Shuo Huang, Fengtao Luo, Meng Xu, Jun Wang, Tingting Wu, Chen Liang, Hangang Chen, Nan Su, Xiaolan Du, Yue Shen, Lin Chen	2016	42
12	A novel FGFR3-binding peptide inhibits FGFR3 signaling and reverses the lethal phenotype of mice mimicking human thanatophoric dysplasia Human Molecular Genetics ·Min Jin, Ying Yu, Huabing Qi, Yangli Xie, Nan Su, Xiaofeng Wang, Qiaoyan Tan, Fengtao Luo, Ying Zhu, Quan Wang, Xiaolan Du, Cory J. Xian, Peng Liu, Haiyang Huang, Yue Shen, Chu‐Xia Deng, Di Chen, Lin Chen	2012	41
13	Generation of Fgfr3 Conditional Knockout Mice International Journal of Biological Sciences ·Nan Su, Xiaoling Xu, Cuiling Li, Qifen He, Ling Zhao, Can Li, Siyu Chen, Fengtao Luo, Lingxian Yi, Xiaolan Du, Haiyang Huang, (unknown), Lin Chen	2010	37
14	FGFR3 induces degradation of BMP type I receptor to regulate skeletal development Biochimica et Biophysica Acta (BBA) - Molecular Cell Research ·Huabing Qi, Min Jin, Yaqi Duan, Xiaolan Du, Yanquan Zhang, Fangli Ren, Yinyin Wang, Qingyun Tian, Xiaofeng Wang, Quan Wang, Ying Zhu, Yangli Xie, Liu C, Xu Cao, Yuji Mishina, Di Chen, Chu‐Xia Deng, Zhijie Chang, Lin Chen	2014	36
15	Fibroblast growth factor receptor 1 regulates the differentiation and activation of osteoclasts through Erk1/2 pathway Biochemical and Biophysical Research Communications ·Xiu‐Min Lu, Nan Su, Jing Yang, Wei Huang, Can Li, Ling Zhao, Qifen He, Xiaolan Du, Yue Shen, Bo Chen, Lin Chen	2009	35
16	Anemonin attenuates osteoarthritis progression through inhibiting the activation of IL‐1β/NF‐κB pathway Journal of Cellular and Molecular Medicine ·Zuqiang Wang, Junlan Huang, Siru Zhou, Fengtao Luo, Wei Xu, Quan Wang, Qiaoyan Tan, Chen Liang, Jun Wang, Hangang Chen, Lin Chen, Yangli Xie, Xiaolan Du	2017	35
17	Loss of Fgfr1 in chondrocytes inhibits osteoarthritis by promoting autophagic activity in temporomandibular joint Journal of Biological Chemistry ·Zuqiang Wang, Junlan Huang, Siru Zhou, Fengtao Luo, Qiaoyan Tan, Xianding Sun, Zhenhong Ni, Hangang Chen, Xiaolan Du, Yangli Xie, Lin Chen	2018	30
18	Cartilage-specific deletion of Alk5 gene results in a progressive osteoarthritis-like phenotype in mice Osteoarthritis and Cartilage ·Qiang Wang, Qiaoyan Tan, Wei Xu, Huabin Qi, Daichao Chen, Siru Zhou, Zhenhong Ni, Lvlin Kuang, Jingyuan Guo, Jianzhao Huang, X.X. Wang, Zhigang Wang, Nan Su, L. Chen, B. Chen, Weiwen Jiang, Yao Gao, H.G. Chen, Xiaolan Du, Yangli Xie, L. Chen	2017	29
19	RECENT RESEARCH ON THE GROWTH PLATE: Advances in fibroblast growth factor signaling in growth plate development and disorders Journal of Molecular Endocrinology ·Yangli Xie, Siru Zhou, Hangang Chen, Xiaolan Du, Lin Chen	2014	28
20	Deletion of FGFR3 in Osteoclast Lineage Cells Results in Increased Bone Mass in Mice by Inhibiting Osteoclastic Bone Resorption Journal of Bone and Mineral Research ·Nan Su, Xiaogang Li, Yubin Tang, Jing Yang, Xuan Wen, Jingyuan Guo, Junzhou Tang, Xiaolan Du, Lin Chen	2016	26

About Xiaolan Du

Xiaolan Du is a scholar working on Molecular Biology, Genetics, Rheumatology, Oncology and Surgery, having authored 51 papers that have together received 1.5k indexed citations. Recurring topics across this work include Fibroblast Growth Factor Research (20 papers), Connective tissue disorders research (11 papers), Osteoarthritis Treatment and Mechanisms (5 papers), Bone Metabolism and Diseases (4 papers), Bone health and treatments (4 papers), TGF-β signaling in diseases (3 papers), Cleft Lip and Palate Research (3 papers) and Craniofacial Disorders and Treatments (3 papers). The work is most often cited by research in Rheumatology (446 citations), Cancer Research (234 citations), Molecular Biology (917 citations), Orthopedics and Sports Medicine (104 citations) and Genetics (347 citations). Xiaolan Du has collaborated with scholars based in China, United States and Australia. Frequent co-authors include Lin Chen, Yangli Xie, Hangang Chen, Nan Su, Siru Zhou, Zhenhong Ni, Junjie Ouyang, Qiaoyan Tan, Cory J. Xian and Liang Kuang. Their work appears in journals such as International Journal of Biological Sciences, Physical review. B., Autophagy, Biochemical and Biophysical Research Communications and Journal of Bone and Mineral Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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