W. David Sedwick

3.4k citations
61 papers · 2.8k · 1 hit paper · h-index 27

Impact in

Papers in

W. David Sedwick

61 papers receiving 2.6k citations

W. David Sedwick's Hit Papers

Biallelic inactivation of hMLH 1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers 1998 · 508 citations
5080+9+18Years since publication100200300400500

Peers

W. David Sedwick
Comparison fields: 5 of 110
  • Pathology and Forensic Medicine 759
  • Cancer Research 595
  • Virology 119
  • Oncology 625
  • Molecular Biology 1.5k
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Citations per year

Countries citing papers authored by W. David Sedwick

Since Specialization
Citations

This map shows the geographic impact of W. David Sedwick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. David Sedwick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. David Sedwick more than expected).

Fields of papers citing papers by W. David Sedwick

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. David Sedwick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. David Sedwick. The network helps show where W. David Sedwick may publish in the future.

Co-authors

The 25 scholars most cited alongside W. David Sedwick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with W. David Sedwick Line = papers co-authored together W. David Sedwick links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 61 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Biallelic inactivation of hMLH 1 by epigenetic gene silencing, a novel mechanism causing human MSI cancers
Hit paper breakdown →
1998508
2
Increased mutation rate at the hprt locus accompanies microsatellite instability in colon cancer.
1995199
3 1972185
4 1984128
5 1998106
6 1967100
7 1974100
8 196596
9 201594
10 198792
11 198790
12 198183
13 200765
14 198465
15
Loss of DNA mismatch repair imparts defective cdc2 signaling and G(2) arrest responses without altering survival after ionizing radiation.
200160
16 200351
17 199551
18 197546
19 198942
20 197541

About W. David Sedwick

W. David Sedwick is a scholar working on Molecular Biology, Pathology and Forensic Medicine, Oncology, Genetics and Epidemiology, having authored 61 papers that have together received 2.8k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (16 papers), DNA and Nucleic Acid Chemistry (15 papers), Genetic factors in colorectal cancer (10 papers), Virology and Viral Diseases (6 papers), Synthesis and Characterization of Heterocyclic Compounds (5 papers), Virus-based gene therapy research (5 papers), Cancer therapeutics and mechanisms (5 papers) and Colorectal Cancer Treatments and Studies (4 papers). The work is most often cited by research in Pathology and Forensic Medicine (759 citations), Cancer Research (595 citations), Virology (119 citations), Oncology (625 citations) and Molecular Biology (1.5k citations). W. David Sedwick has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Martina Veigl, David Korn, Teresa Shu-Fong Wang, Sanford D. Markowitz, Lawrence C. Sowers, Barbara Ramsay Shaw, James Lutterbaugh, Paul Modrich, Guo‐Min Li and Antti Vaheri. Their work appears in journals such as Journal of Biological Chemistry, Mutation research. Fundamental and molecular mechanisms of mutagenesis, Experimental Biology and Medicine, Molecular Pharmacology and Oncogene.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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