Ute Boon

1.4k citations
6 papers · 713 · h-index 4

Impact in

  • Oncology top 5%
    • PARP inhibition in cancer therapy
    • Cancer-related Molecular Pathways
    • DNA Repair Mechanisms
    • CRISPR and Genetic Engineering

Papers in

    • DNA Repair Mechanisms 3
    • Genomics and Chromatin Dynamics 2
    • Epigenetics and DNA Methylation 1
    • RNA modifications and cancer 1
    • Protein Tyrosine Phosphatases 1
    • PARP inhibition in cancer therapy 2

Ute Boon

5 papers receiving 705 citations

Peers

Ute Boon
Comparison fields: 5 of 44
  • Oncology 506
  • Molecular Biology 603
  • Reproductive Medicine 57
  • Genetics 178
  • Cancer Research 85
Replace Ewa Gogola with:
Ewa Gogola Netherlands
Niraj Joshi Canada
Benjamin Primack United States
Anbazhagan Rajendran United States
Hatice Yücel Netherlands
Dongxue Su China
Kamakoti P. Bhat United States
Giuseppe Leuzzi United States
Rinske Drost Netherlands
Ute Boon relative to Ewa Gogola Netherlands Ewa Gogola's profile →
Citations per field
00.5×4.9×
Ewa Gogola · 1×
Citations per year

Countries citing papers authored by Ute Boon

Since Specialization
Citations

This map shows the geographic impact of Ute Boon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ute Boon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ute Boon more than expected).

Fields of papers citing papers by Ute Boon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ute Boon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ute Boon. The network helps show where Ute Boon may publish in the future.

Co-authors

The 25 scholars most cited alongside Ute Boon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ute Boon Line = papers co-authored together Ute Boon links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 2012388
2 2011197
3 201593
4 201833
5 20151
6 20161

About Ute Boon

Ute Boon is a scholar working on Molecular Biology, Oncology, Cell Biology, Pathology and Forensic Medicine and Genetics, having authored 6 papers that have together received 713 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (3 papers), Microtubule and mitosis dynamics (3 papers), PARP inhibition in cancer therapy (2 papers), Genomics and Chromatin Dynamics (2 papers), Epigenetics and DNA Methylation (1 paper), RNA modifications and cancer (1 paper), Protein Tyrosine Phosphatases (1 paper) and BRCA gene mutations in cancer (1 paper). The work is most often cited by research in Oncology (506 citations), Molecular Biology (603 citations), Reproductive Medicine (57 citations), Genetics (178 citations) and Cancer Research (85 citations). Ute Boon has collaborated with scholars based in Netherlands, United Kingdom and Germany. Frequent co-authors include Jos Jonkers, Sven Rottenberg, Ellen Wientjens, Rinske Drost, Liesbeth van Deemter, Shridar Ganesan, Piet Borst, James H. Doroshow, Janneke E. Jaspers and Jiuping Ji. Their work appears in journals such as Annals of Oncology, Cancer Discovery, British Journal of Cancer, Cancer Cell and Geburtshilfe und Frauenheilkunde.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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