Thomas Wa
Impact in
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- Atherosclerosis and Cardiovascular Diseases
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- Cell Adhesion Molecules Research
Papers in
- Surgery 5
- Transplantation: Methods and Outcomes 2
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- Cardiac Valve Diseases and Treatments 2
- Co-authors
- Lee Kt (12 shared papers)H. Imai (4 shared papers)F. Goodale (4 shared papers)Rose M. Jones (1 shared paper)E.S. Morrison (1 shared paper)Lee Sk (1 shared paper)K. Janakidevi (2 shared papers)Hartroft Ws (1 shared paper)
- Journals
- PubMed (33 papers)
- Partner nations
- United States
In The Last Decade
Thomas Wa
32 papers receiving 344 citations
Peers
Comparison fields: 5 of 71
- Immunology 110
- Immunology and Allergy 30
- Cardiology and Cardiovascular Medicine 107
- Biochemistry 17
- Pulmonary and Respiratory Medicine 72
Countries citing papers authored by Thomas Wa
This map shows the geographic impact of Thomas Wa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Wa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Wa more than expected).
Fields of papers citing papers by Thomas Wa
This network shows the impact of papers produced by Thomas Wa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Wa. The network helps show where Thomas Wa may publish in the future.
Co-authors
The 16 scholars most cited alongside Thomas Wa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Biology of disease. Atherosclerosis as a hyperplastic and/or neoplastic process. | 1983 | 90 |
| 2 | PRODUCTION OF EARLY ATHEROSCLEROTIC LESIONS IN RATS CHARACTERIZED BY PROLIFERATION OF "MODIFIED SMOOTH MUSCLE CELLS". | 1963 | 72 |
| 3 | Increased mitotic activity in aortas of swine after three days of cholesterol feeding. | 1969 | 60 |
| 4 | Ultrastructural features of aortic cells in mitosis in control and cholesterol-fed swine. | 1970 | 32 |
| 5 | Primary pulmonary arterial disease; observations with special reference to medial thickening of small arteries and arterioles. | 1954 | 25 |
| 6 | The media of small muscular pulmonary arteries in mitral stenosis. | 1955 | 19 |
| 7 | Population dynamics of arterial smooth-muscle cells. II. In vivo inhibition of entry into mitosis of swine arterial smooth-muscle cells by aortic tissue extracts. | 1973 | 13 |
| 8 | Thromboembolism, pulmonary arteriosclerosis, and fatty meals; an experimental study of the effect of intermittent fatty meals on thromboembolic-induced pulmonary arteriosclerosis in rabbits. | 1956 | 10 |
| 9 | Mitral stenosis and pulmonary arteriosclerosis; correlation of pulmonary arteriosclerosis, right ventricular hypertrophy, and thromboembolism in autopsied patients who died with mitral stenosis. | 1956 | 8 |
| 10 | Myocardial infarction in women. A study of autopsy populations. | 1960 | 7 |
| 11 | Conformations of cyclic and cylindrical peptides. | 1971 | 7 |
| 12 | Pathogenesis of atherosclerosis and myocardial infarction. | 1959 | 7 |
| 13 | Effect of intravenous fibrinolytic enzymes on the vegetations of experimental bacterial endocarditis. | 1958 | 6 |
| 14 | Smooth muscle and endothelial cell deaths in atherogenesis studied by autoradiography. | 1977 | 6 |
| 15 | Thromboembolism and experimental systemic arteriosclerosis. | 1957 | 6 |
| 16 | Comparable survival of intra-species and cross-species primate cardiac transplants. | 1987 | 5 |
| 17 | Cyclosporine increases rat heart weight in heterotopic transplants. | 1987 | 5 |
| 18 | Survival of rats with induced congenital cardiovascular anomalies. | 1957 | 5 |
| 19 | Dietary lipids, thrombosis, and clot-lysis. Chemicopathologic studies. III. The effect on blood lipid levels in the rat of purified linoleic and oleic acids substituted in or added to thrombogenic diets. | 1961 | 4 |
| 20 | DIETARY LIPIDS AND THROMBOSIS: IN VITRO STUDIES OF MULTIPLE COAGULATION FACTORS IN RATS FED THROMBOGENIC AND NONTHROMBOGENIC DIETS, WITH SPECIAL REFERENCE TO POTENTIAL "HYPERCOAGULABLE" STATES. | 1963 | 4 |
About Thomas Wa
Thomas Wa is a scholar working on Surgery, Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine, Epidemiology and Physiology, having authored 33 papers that have together received 419 indexed citations. Recurring topics across this work include Pulmonary Hypertension Research and Treatments (4 papers), Atherosclerosis and Cardiovascular Diseases (3 papers), Diet and metabolism studies (3 papers), Cancer, Lipids, and Metabolism (2 papers), Cardiac Valve Diseases and Treatments (2 papers), Diet, Metabolism, and Disease (2 papers), Transplantation: Methods and Outcomes (2 papers) and Congenital Heart Disease Studies (2 papers). The work is most often cited by research in Immunology (110 citations), Immunology and Allergy (30 citations), Cardiology and Cardiovascular Medicine (107 citations), Biochemistry (17 citations) and Pulmonary and Respiratory Medicine (72 citations). Thomas Wa has collaborated with scholars based in United States. Frequent co-authors include Lee Kt, H. Imai, F. Goodale, Rose M. Jones, E.S. Morrison, Lee Sk, K. Janakidevi, Hartroft Ws, K Reemtsma and C. Smith. Their work appears in journals such as PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.