Thomas Ried

372 papers receiving 27.4k citations

Thomas Ried's Hit Papers

SIRT6 is a histone H3 lysine 9 deacetylase that modulates telomeric chromatin 2008 · 857 citations
8570+10+20Years since publication4008001.2k

Peers

Thomas Ried
Comparison fields: 5 of 168
  • Cancer Research 5.5k
  • Geriatrics and Gerontology 1.0k
  • Oncology 6.5k
  • Molecular Biology 16.4k
  • Genetics 5.8k
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Citations per year

Countries citing papers authored by Thomas Ried

Since Specialization
Citations

This map shows the geographic impact of Thomas Ried's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Ried with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Ried more than expected).

Fields of papers citing papers by Thomas Ried

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Ried. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Ried. The network helps show where Thomas Ried may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas Ried, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Ried Line = papers co-authored together Thomas Ried links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 377 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Multicolor Spectral Karyotyping of Human Chromosomes
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19961314
2
Atm-Deficient Mice: A Paradigm of Ataxia Telangiectasia
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19961211
3
SIRT6 is a histone H3 lysine 9 deacetylase that modulates telomeric chromatin
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2008857
4
Impaired DNA Damage Response, Genome Instability, and Tumorigenesis in SIRT1 Mutant Mice
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2008638
5
Conditional mutation of Brca1 in mammary epithelial cells results in blunted ductal morphogenesis and tumour formation
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1999599
6
From Silencing to Gene Expression
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2004569
7
Frequent translocation t(4;14)(p16.3;q32.3) in multiple myeloma is associated with increased expression and activating mutations of fibroblast growth factor receptor 3
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1997527
8 2000457
9 2005456
10 2003448
11
AID is required to initiate Nbs1/γ-H2AX focus formation and mutations at sites of class switching
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2001410
12 2007409
13 1993392
14 1996388
15 2013333
16 1992306
17 1998304
18 1997282
19 1998274
20 2005269

About Thomas Ried

Thomas Ried is a scholar working on Molecular Biology, Genetics, Oncology, Cancer Research and Pathology and Forensic Medicine, having authored 377 papers that have together received 27.9k indexed citations. Recurring topics across this work include Genomic variations and chromosomal abnormalities (108 papers), Cancer Genomics and Diagnostics (71 papers), Genetic factors in colorectal cancer (51 papers), Chromosomal and Genetic Variations (44 papers), Genomics and Chromatin Dynamics (42 papers), DNA Repair Mechanisms (40 papers), Cancer-related Molecular Pathways (36 papers) and Microtubule and mitosis dynamics (21 papers). The work is most often cited by research in Cancer Research (5.5k citations), Geriatrics and Gerontology (1.0k citations), Oncology (6.5k citations), Molecular Biology (16.4k citations) and Genetics (5.8k citations). Thomas Ried has collaborated with scholars based in United States, Germany and Sweden. Frequent co-authors include Evelin Schröck, Michael J. Difilippantonio, Hesed Padilla‐Nash, Stanislas du Manoir, Β. Michael Ghadimi, Gert Auer, Anthony Wynshaw‐Boris, André Nussenzweig, Jordi Camps and Marek Liyanage. Their work appears in journals such as Genes Chromosomes and Cancer, Cancer Research, Oncogene, International Journal of Cancer and Blood.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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