Thomas Kryza

1.1k citations
33 papers · 657 · h-index 16

Impact in

  • Genetics top 10%
    • Coagulation, Bradykinin, Polyphosphates, and Angioedema
    • Protease and Inhibitor Mechanisms

Papers in

    • Peptidase Inhibition and Analysis 8
    • HER2/EGFR in Cancer Research 3
    • Coagulation, Bradykinin, Polyphosphates, and Angioedema 12

Thomas Kryza

32 papers receiving 650 citations

Peers

Thomas Kryza
Comparison fields: 5 of 82
  • Genetics 143
  • Cancer Research 160
  • Oncology 268
  • Immunology and Allergy 46
  • Hematology 48
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Citations per field
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Citations per year

Countries citing papers authored by Thomas Kryza

Since Specialization
Citations

This map shows the geographic impact of Thomas Kryza's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Kryza with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Kryza more than expected).

Fields of papers citing papers by Thomas Kryza

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Kryza. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Kryza. The network helps show where Thomas Kryza may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas Kryza, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Kryza Line = papers co-authored together Thomas Kryza links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 33 papers — load more, or switch the sort, to bring in the rest.

#Work
1 201573
2 201866
3 202248
4 202145
5 202337
6 201637
7 202033
8 201931
9 201229
10 201329
11 202129
12 202422
13 201721
14 202121
15 202116
16 201916
17 201214
18 201813
19 201913
20 202311

About Thomas Kryza

Thomas Kryza is a scholar working on Oncology, Genetics, Cancer Research, Radiology, Nuclear Medicine and Imaging and Pulmonary and Respiratory Medicine, having authored 33 papers that have together received 657 indexed citations. Recurring topics across this work include Coagulation, Bradykinin, Polyphosphates, and Angioedema (12 papers), Radiopharmaceutical Chemistry and Applications (8 papers), Protease and Inhibitor Mechanisms (8 papers), Peptidase Inhibition and Analysis (8 papers), Prostate Cancer Treatment and Research (7 papers), HER2/EGFR in Cancer Research (3 papers), Blood Coagulation and Thrombosis Mechanisms (3 papers) and Medical Imaging Techniques and Applications (2 papers). The work is most often cited by research in Genetics (143 citations), Cancer Research (160 citations), Oncology (268 citations), Immunology and Allergy (46 citations) and Hematology (48 citations). Thomas Kryza has collaborated with scholars based in Australia, France and United Kingdom. Frequent co-authors include Judith A. Clements, Jyotsna Batra, Nathalie Heuzé‐Vourc'h, John D. Hooper, Lakmali M. Silva, Yaowu He, Samaneh Farashi, Nicholas Lyons, Daniela Loessner and Nathalie Bock. Their work appears in journals such as Nature reviews. Cancer, Scientific Reports, Molecular Oncology, Biological Chemistry and Journal of Clinical Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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