Thomas B. Shima

686 citations
9 papers · 587 · h-index 6

Impact in

Papers in

    • HER2/EGFR in Cancer Research 3
    • Cancer Cells and Metastasis 2
    • PARP inhibition in cancer therapy 2
    • Cancer therapeutics and mechanisms 1

Thomas B. Shima

9 papers receiving 572 citations

Peers

Thomas B. Shima
Comparison fields: 5 of 76
  • Immunology and Allergy 95
  • Cancer Research 126
  • Oncology 205
  • Cell Biology 109
  • Molecular Biology 310
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Citations per year

Countries citing papers authored by Thomas B. Shima

Since Specialization
Citations

This map shows the geographic impact of Thomas B. Shima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas B. Shima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas B. Shima more than expected).

Fields of papers citing papers by Thomas B. Shima

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas B. Shima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas B. Shima. The network helps show where Thomas B. Shima may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas B. Shima, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas B. Shima Line = papers co-authored together Thomas B. Shima links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 1992447
2
ICI 164,384, a pure antagonist of estrogen-stimulated MCF-7 cell proliferation and invasiveness.
198935
3 199529
4
Carzinophilin, a new tumor inhibitory substance produced by streptomyces. I.
195428
5
Differential regulation of growth and invasiveness of MCF-7 breast cancer cells by antiestrogens.
198824
6
Studies on antitumor activity of mitomycin.
195715
7 19944
8 19904
9
Regulation of invasiveness of human breast cancer cell lines in vitro.
19881

About Thomas B. Shima

Thomas B. Shima is a scholar working on Oncology, Molecular Biology, Genetics, Cardiology and Cardiovascular Medicine and Toxicology, having authored 9 papers that have together received 587 indexed citations. Recurring topics across this work include HER2/EGFR in Cancer Research (3 papers), Estrogen and related hormone effects (2 papers), Cancer Cells and Metastasis (2 papers), PARP inhibition in cancer therapy (2 papers), Synthesis and Biological Activity (1 paper), Bioactive Compounds and Antitumor Agents (1 paper), Toxin Mechanisms and Immunotoxins (1 paper) and Cancer therapeutics and mechanisms (1 paper). The work is most often cited by research in Immunology and Allergy (95 citations), Cancer Research (126 citations), Oncology (205 citations), Cell Biology (109 citations) and Molecular Biology (310 citations). Thomas B. Shima has collaborated with scholars based in United States. Frequent co-authors include Robert B. Dickson, Erik W. Thompson, Marc E. Lippman, Gerhard Zugmaier, George R. Martin, Jeffrey A. Torri, Connie L. Sommers, Robert Clarke, Steven Donahue and Soonmyoung Paik. Their work appears in journals such as Biochemical and Biophysical Research Communications, Journal of Cellular Physiology, Annals of the New York Academy of Sciences, Journal of Investigative Dermatology and PubMed.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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