Thilo Mengling

880 citations
15 papers · 617 · h-index 7

Impact in

  • Hematology top 5%
    • Hematopoietic Stem Cell Transplantation
    • interferon and immune responses
    • Immune Response and Inflammation
    • Immunotherapy and Immune Responses

Papers in

    • Hematopoietic Stem Cell Transplantation 9
    • Acute Myeloid Leukemia Research 2
    • Cell death mechanisms and regulation 4
    • Ubiquitin and proteasome pathways 2

Thilo Mengling

13 papers receiving 602 citations

Peers

Thilo Mengling
Comparison fields: 5 of 62
  • Hematology 141
  • Immunology 196
  • Cancer Research 134
  • Transplantation 16
  • Oncology 134
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Citations per field
00.5×1.5×1.8×
Steven Schutt · 1×
Citations per year

Countries citing papers authored by Thilo Mengling

Since Specialization
Citations

This map shows the geographic impact of Thilo Mengling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thilo Mengling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thilo Mengling more than expected).

Fields of papers citing papers by Thilo Mengling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thilo Mengling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thilo Mengling. The network helps show where Thilo Mengling may publish in the future.

Co-authors

The 25 scholars most cited alongside Thilo Mengling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thilo Mengling Line = papers co-authored together Thilo Mengling links everyone, so they are left out of the graph.

All Works

15 of 15 papers shown
#Work
1
Regulation of tumor necrosis factor-related apoptosis-inducing ligand sensitivity in primary and transformed human keratinocytes.
2000265
2 2009164
3 200399
4 200036
5 201717
6 201011
7 20168
8 20205
9 20165
10 20033
11 20202
12 20231
13 20161
14 20150
15 20250

About Thilo Mengling

Thilo Mengling is a scholar working on Hematology, Molecular Biology, Physiology, Immunology and Oncology, having authored 15 papers that have together received 617 indexed citations. Recurring topics across this work include Hematopoietic Stem Cell Transplantation (9 papers), Cell death mechanisms and regulation (4 papers), Biomedical Ethics and Regulation (4 papers), Acute Myeloid Leukemia Research (2 papers), Ubiquitin and proteasome pathways (2 papers), Immunotherapy and Immune Responses (2 papers), Acute Lymphoblastic Leukemia research (1 paper) and NF-κB Signaling Pathways (1 paper). The work is most often cited by research in Hematology (141 citations), Immunology (196 citations), Cancer Research (134 citations), Transplantation (16 citations) and Oncology (134 citations). Thilo Mengling has collaborated with scholars based in Germany, United States and Canada. Frequent co-authors include Manfred Neumann, Henning Walczak, M. Leverkus, Charles T. Rauch, P H Krammer, E B Bröcker, Gerhard Ehninger, Alexander H. Schmidt, Kristina Hölig and Claudia Rutt. Their work appears in journals such as Blood, Molecular and Cellular Biology, Transfusion, Cell Death and Differentiation and Bone Marrow Transplantation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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