Takehiro Ogata

87 papers receiving 2.8k citations

Takehiro Ogata's Hit Papers

Cytosolic p53 inhibits Parkin-mediated mitophagy and promotes mitochondrial dysfunction in the mouse heart 2013 · 449 citations
4490+4+8Years since publication100200300400

Peers

Takehiro Ogata
Comparison fields: 5 of 112
  • Cardiology and Cardiovascular Medicine 569
  • Environmental Chemistry 287
  • Cell Biology 396
  • Molecular Biology 1.3k
  • Biophysics 93
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Citations per year

Countries citing papers authored by Takehiro Ogata

Since Specialization
Citations

This map shows the geographic impact of Takehiro Ogata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takehiro Ogata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takehiro Ogata more than expected).

Fields of papers citing papers by Takehiro Ogata

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takehiro Ogata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takehiro Ogata. The network helps show where Takehiro Ogata may publish in the future.

Co-authors

The 25 scholars most cited alongside Takehiro Ogata, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Takehiro Ogata Line = papers co-authored together Takehiro Ogata links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 93 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Cytosolic p53 inhibits Parkin-mediated mitophagy and promotes mitochondrial dysfunction in the mouse heart
Hit paper breakdown →
2013449
2 2014190
3 2008162
4 2004141
5 1987135
6 2017132
7 2002104
8 2004102
9 200899
10 200873
11 198967
12 198563
13 200856
14 201453
15 201052
16 200250
17 201348
18 200847
19 200544
20 201141

About Takehiro Ogata

Takehiro Ogata is a scholar working on Molecular Biology, Cell Biology, Cardiology and Cardiovascular Medicine, Pulmonary and Respiratory Medicine and Physiology, having authored 93 papers that have together received 2.8k indexed citations. Recurring topics across this work include Caveolin-1 and cellular processes (12 papers), Marine Toxins and Detection Methods (9 papers), Adipose Tissue and Metabolism (6 papers), Pulmonary Hypertension Research and Treatments (6 papers), Peroxisome Proliferator-Activated Receptors (5 papers), Marine and coastal ecosystems (5 papers), Cardiovascular Function and Risk Factors (5 papers) and Cardiovascular Disease and Adiposity (5 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (569 citations), Environmental Chemistry (287 citations), Cell Biology (396 citations), Molecular Biology (1.3k citations) and Biophysics (93 citations). Takehiro Ogata has collaborated with scholars based in Japan, United States and China. Frequent co-authors include Tomomi Ueyama, Satoaki Matoba, Masaaki Kodama, Koji Ikeda, Atsushi Hoshino, Yoshifumi Okawa, Makoto Ariyoshi, Eri Iwai‐Kanai, Yoko Irukayama‐Tomobe and Takashi Miyauchi. Their work appears in journals such as Biochemical and Biophysical Research Communications, Marine Ecology Progress Series, Cells, American Journal of Physiology-Heart and Circulatory Physiology and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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