Stephen E. Kaiser

2.3k citations
24 papers · 1.7k · h-index 18

Impact in

    • Endoplasmic Reticulum Stress and Disease
    • Cellular transport and secretion
    • Ubiquitin and proteasome pathways
    • RNA modifications and cancer
    • Lipid Membrane Structure and Behavior

Papers in

    • Ubiquitin and proteasome pathways 11
    • Epigenetics and DNA Methylation 4
    • Lipid Membrane Structure and Behavior 3
    • Autophagy in Disease and Therapy 8

Stephen E. Kaiser

24 papers receiving 1.6k citations

Peers

Stephen E. Kaiser
Comparison fields: 5 of 111
  • Cell Biology 548
  • Molecular Biology 1.2k
  • Physiology 71
  • Epidemiology 461
  • Aging 16
Replace Heeseon An with:
Heeseon An United States
Brenda Kostelecky United Kingdom
Jimi Wills United Kingdom
Boaz Inbal Israel
Chad J. Miller United States
Mogens M. Nielsen Denmark
Shuliang Chen United States
Christopher Berndsen United States
Sarah Hanrahan United Kingdom
Mark H. Watson United States
Stephen E. Kaiser relative to Heeseon An United States Heeseon An's profile →
Citations per field
00.5×5.8×
Heeseon An · 1×
Citations per year

Countries citing papers authored by Stephen E. Kaiser

Since Specialization
Citations

This map shows the geographic impact of Stephen E. Kaiser's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen E. Kaiser with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen E. Kaiser more than expected).

Fields of papers citing papers by Stephen E. Kaiser

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen E. Kaiser. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen E. Kaiser. The network helps show where Stephen E. Kaiser may publish in the future.

Co-authors

The 25 scholars most cited alongside Stephen E. Kaiser, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Stephen E. Kaiser Line = papers co-authored together Stephen E. Kaiser links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 24 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2005223
2 2011193
3 2010150
4 2012142
5 2011137
6 2015126
7 201798
8 201289
9 201188
10 201265
11 202057
12 200152
13 201648
14 200742
15 201334
16 197633
17 201120
18 201318
19 201212
20 200810

About Stephen E. Kaiser

Stephen E. Kaiser is a scholar working on Molecular Biology, Epidemiology, Cell Biology, Physiology and Physiology, having authored 24 papers that have together received 1.7k indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (11 papers), Autophagy in Disease and Therapy (8 papers), Cellular transport and secretion (4 papers), Epigenetics and DNA Methylation (4 papers), Endoplasmic Reticulum Stress and Disease (3 papers), Adenosine and Purinergic Signaling (3 papers), Lipid Membrane Structure and Behavior (3 papers) and Genetics and Neurodevelopmental Disorders (2 papers). The work is most often cited by research in Cell Biology (548 citations), Molecular Biology (1.2k citations), Physiology (71 citations), Epidemiology (461 citations) and Aging (16 citations). Stephen E. Kaiser has collaborated with scholars based in United States, Germany and United Kingdom. Frequent co-authors include Ron R. Kopito, Thomas A. Shaler, Brigit E. Riley, Axel T. Brünger, Amy Reilein, Peter Walter, Jason H. Brickner, Brenda A. Schulman, Christopher H. Becker and Mark S. Hipp. Their work appears in journals such as Molecular Cell, The Journal of Cell Biology, EMBO Reports, Autophagy and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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