Stanley D. Handmaker

538 citations
11 papers · 429 · h-index 8

Impact in

Papers in

    • CRISPR and Genetic Engineering 2
    • RNA and protein synthesis mechanisms 2
    • DNA Repair Mechanisms 2
    • Virus-based gene therapy research 1

Stanley D. Handmaker

11 papers receiving 370 citations

Peers

Stanley D. Handmaker
Comparison fields: 5 of 74
  • Pediatrics, Perinatology and Child Health 188
  • Obstetrics and Gynecology 51
  • Endocrine and Autonomic Systems 24
  • Developmental Biology 6
  • Cognitive Neuroscience 50
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Citations per field
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Citations per year

Countries citing papers authored by Stanley D. Handmaker

Since Specialization
Citations

This map shows the geographic impact of Stanley D. Handmaker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stanley D. Handmaker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stanley D. Handmaker more than expected).

Fields of papers citing papers by Stanley D. Handmaker

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stanley D. Handmaker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stanley D. Handmaker. The network helps show where Stanley D. Handmaker may publish in the future.

Co-authors

The 14 scholars most cited alongside Stanley D. Handmaker, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Stanley D. Handmaker Line = papers co-authored together Stanley D. Handmaker links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 1995190
2 199553
3 197051
4
The satellited chromosomes of man with reference to the marfan syndrome.
196329
5 197128
6
Dyssegmental dwarfism: a new syndrome of lethal dwarfism.
197725
7 197022
8 197120
9 19675
10 19755
11 19711

About Stanley D. Handmaker

Stanley D. Handmaker is a scholar working on Molecular Biology, Genetics, Pediatrics, Perinatology and Child Health, Infectious Diseases and Surgery, having authored 11 papers that have together received 429 indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (2 papers), RNA and protein synthesis mechanisms (2 papers), DNA Repair Mechanisms (2 papers), Prenatal Substance Exposure Effects (1 paper), Neonatal and fetal brain pathology (1 paper), Virus-based gene therapy research (1 paper), Prenatal Screening and Diagnostics (1 paper) and Autoimmune and Inflammatory Disorders Research (1 paper). The work is most often cited by research in Pediatrics, Perinatology and Child Health (188 citations), Obstetrics and Gynecology (51 citations), Endocrine and Autonomic Systems (24 citations), Developmental Biology (6 citations) and Cognitive Neuroscience (50 citations). Stanley D. Handmaker has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Piyadasa Kodituwakku, Susan K. Cutler, Nancy S. Handmaker, M. E. Bramwell, Kristina T. Ciesielski, Ronald J. Prince, Richard Harris, John W. Graef, Robert J. Gorlin and James A. Campbell. Their work appears in journals such as Experimental Cell Research, Life Sciences, Nature, Neuropsychologia and Alcoholism Clinical and Experimental Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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