Seth D. Kasowitz
Impact in
- Cancer Research top 10%
- Cancer-related molecular mechanisms research
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- RNA modifications and cancer
- Cancer-related gene regulation
- RNA Research and Splicing
- RNA and protein synthesis mechanisms
- Epigenetics and DNA Methylation
Papers in
-
- CRISPR and Genetic Engineering 3
- DNA Repair Mechanisms 2
- RNA modifications and cancer 2
- Epigenetics and DNA Methylation 1
- Pluripotent Stem Cells Research 1
- Genetics 2
- Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities 1
- Genetics and Neurodevelopmental Disorders 1
- Genetic Syndromes and Imprinting 1
- Co-authors
- P. Jeremy Wang (4 shared papers)N. Adrian Leu (3 shared papers)Jun Ma (1 shared paper)Yang Xu (1 shared paper)Brian D. Gregory (1 shared paper)Richard M. Schultz (1 shared paper)Stephen J. Anderson (1 shared paper)Mengcheng Luo (2 shared papers)
- Journals
- PLoS Genetics (2 papers)BMC Genomics (1 paper)Epigenetics & Chromatin (1 paper)Cell Reports (1 paper)The CRISPR Journal (1 paper)
- Partner nations
- United StatesChinaSwitzerland
In The Last Decade
Seth D. Kasowitz
7 papers receiving 516 citations
Seth D. Kasowitz's Hit Papers
Peers
Comparison fields: 5 of 40
- Cancer Research 211
- Molecular Biology 476
- Genetics 37
- Electrical and Electronic Engineering 69
- Reproductive Medicine 9
Countries citing papers authored by Seth D. Kasowitz
This map shows the geographic impact of Seth D. Kasowitz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seth D. Kasowitz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seth D. Kasowitz more than expected).
Fields of papers citing papers by Seth D. Kasowitz
This network shows the impact of papers produced by Seth D. Kasowitz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seth D. Kasowitz. The network helps show where Seth D. Kasowitz may publish in the future.
Co-authors
The 25 scholars most cited alongside Seth D. Kasowitz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Nuclear m6A reader YTHDC1 regulates alternative polyadenylation and splicing during mouse oocyte development Hit paper breakdown → | 2018 | 429 |
| 2 | 2021 | 32 | |
| 3 | 2013 | 28 | |
| 4 | 2018 | 15 | |
| 5 | 2017 | 9 | |
| 6 | 2023 | 3 | |
| 7 | 2013 | 1 |
About Seth D. Kasowitz
Seth D. Kasowitz is a scholar working on Molecular Biology, Genetics, Business and International Management, Public Health, Environmental and Occupational Health and Insect Science, having authored 7 papers that have together received 517 indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (3 papers), DNA Repair Mechanisms (2 papers), RNA modifications and cancer (2 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (1 paper), Epigenetics and DNA Methylation (1 paper), Genetics and Neurodevelopmental Disorders (1 paper), Genetic Syndromes and Imprinting (1 paper) and Pluripotent Stem Cells Research (1 paper). The work is most often cited by research in Cancer Research (211 citations), Molecular Biology (476 citations), Genetics (37 citations), Electrical and Electronic Engineering (69 citations) and Reproductive Medicine (9 citations). Seth D. Kasowitz has collaborated with scholars based in United States, China and Switzerland. Frequent co-authors include P. Jeremy Wang, N. Adrian Leu, Jun Ma, Yang Xu, Brian D. Gregory, Richard M. Schultz, Stephen J. Anderson, Mengcheng Luo, Rachel J. O’Neill and Rong Liu. Their work appears in journals such as PLoS Genetics, BMC Genomics, Epigenetics & Chromatin, Cell Reports and The CRISPR Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.