Sara Lööf
Impact in
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- Developmental Biology and Gene Regulation
- Muscle Physiology and Disorders
- Pluripotent Stem Cells Research
- Congenital heart defects research
- Planarian Biology and Electrostimulation
Papers in
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- Muscle Physiology and Disorders 3
- Developmental Biology and Gene Regulation 2
- RNA Research and Splicing 1
- DNA Repair Mechanisms 1
- Ubiquitin and proteasome pathways 1
- Surgery 2
- Co-authors
- András Simon (5 shared papers)Jamie I. Morrison (2 shared papers)Pingping He (2 shared papers)Helge Dalen (1 shared paper)Alexei Terman (1 shared paper)Gustavo A. Nader (1 shared paper)Heng Wang (1 shared paper)Helen M. Blau (1 shared paper)
- Journals
- Cancer Research (2 papers)Nature Communications (1 paper)Developmental Dynamics (1 paper)Cell Reports (1 paper)European Journal of Cell Biology (1 paper)
- Partner nations
- SwedenSingaporeUnited States
In The Last Decade
Sara Lööf
10 papers receiving 380 citations
Peers
Comparison fields: 5 of 79
- Aging 14
- Molecular Biology 281
- Developmental Biology 7
- Cell Biology 46
- Physiology 12
Countries citing papers authored by Sara Lööf
This map shows the geographic impact of Sara Lööf's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara Lööf with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara Lööf more than expected).
Fields of papers citing papers by Sara Lööf
This network shows the impact of papers produced by Sara Lööf. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara Lööf. The network helps show where Sara Lööf may publish in the future.
Co-authors
The 25 scholars most cited alongside Sara Lööf, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 188 | |
| 2 | 2004 | 78 | |
| 3 | 2015 | 42 | |
| 4 | 2019 | 23 | |
| 5 | 2010 | 23 | |
| 6 | 2007 | 15 | |
| 7 | 2006 | 7 | |
| 8 | 2024 | 3 | |
| 9 | 2017 | 2 | |
| 10 | 2016 | 1 |
About Sara Lööf
Sara Lööf is a scholar working on Molecular Biology, Surgery, Oncology, Cell Biology and Pulmonary and Respiratory Medicine, having authored 10 papers that have together received 382 indexed citations. Recurring topics across this work include Muscle Physiology and Disorders (3 papers), Developmental Biology and Gene Regulation (2 papers), RNA Research and Splicing (1 paper), DNA Repair Mechanisms (1 paper), Electrospun Nanofibers in Biomedical Applications (1 paper), Ubiquitin and proteasome pathways (1 paper), Virus-based gene therapy research (1 paper) and Graphene and Nanomaterials Applications (1 paper). The work is most often cited by research in Aging (14 citations), Molecular Biology (281 citations), Developmental Biology (7 citations), Cell Biology (46 citations) and Physiology (12 citations). Sara Lööf has collaborated with scholars based in Sweden, Singapore and United States. Frequent co-authors include András Simon, Jamie I. Morrison, Pingping He, Helge Dalen, Alexei Terman, Gustavo A. Nader, Heng Wang, Helen M. Blau, David Drechsel and Joseph D. Bruton. Their work appears in journals such as Cancer Research, Nature Communications, Developmental Dynamics, Cell Reports and European Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.