Ruth Eichner
Impact in
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- Ubiquitin and proteasome pathways
- Protein Degradation and Inhibitors
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- Cancer-related Molecular Pathways
- Peptidase Inhibition and Analysis
Papers in
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- Protein Degradation and Inhibitors 5
- Ubiquitin and proteasome pathways 2
- Ion Transport and Channel Regulation 1
- Oncology 4
- Drug Transport and Resistance Mechanisms 2
- Co-authors
- Florian Bassermann (5 shared papers)Michele Pagano (2 shared papers)Arno Müllbacher (1 shared paper)Paul Waring (1 shared paper)Giulio Superti‐Furga (2 shared papers)Enrico Girardi (2 shared papers)Vanesa Fernández‐Sáiz (2 shared papers)Bianca‐Sabrina Targosz (2 shared papers)
- Journals
- ACS Chemical Biology (1 paper)Microbiology (1 paper)Blood (1 paper)Nature Cell Biology (1 paper)Cell Host & Microbe (1 paper)
- Partner nations
- GermanyAustriaUnited States
In The Last Decade
Ruth Eichner
9 papers receiving 569 citations
Peers
Comparison fields: 5 of 79
- Molecular Biology 369
- Oncology 120
- Cancer Research 63
- Cell Biology 74
- Infectious Diseases 73
Countries citing papers authored by Ruth Eichner
This map shows the geographic impact of Ruth Eichner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ruth Eichner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ruth Eichner more than expected).
Fields of papers citing papers by Ruth Eichner
This network shows the impact of papers produced by Ruth Eichner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ruth Eichner. The network helps show where Ruth Eichner may publish in the future.
Co-authors
The 25 scholars most cited alongside Ruth Eichner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2013 | 207 | |
| 2 | 1985 | 120 | |
| 3 | 2012 | 79 | |
| 4 | 2018 | 64 | |
| 5 | 2012 | 49 | |
| 6 | 2018 | 35 | |
| 7 | 2018 | 20 | |
| 8 | 2021 | 7 | |
| 9 | 2019 | 1 |
About Ruth Eichner
Ruth Eichner is a scholar working on Molecular Biology, Oncology, Hematology, Infectious Diseases and Small Animals, having authored 9 papers that have together received 582 indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (5 papers), Multiple Myeloma Research and Treatments (4 papers), Ubiquitin and proteasome pathways (2 papers), Drug Transport and Resistance Mechanisms (2 papers), Cancer Mechanisms and Therapy (1 paper), Ion Transport and Channel Regulation (1 paper), Antifungal resistance and susceptibility (1 paper) and Amino Acid Enzymes and Metabolism (1 paper). The work is most often cited by research in Molecular Biology (369 citations), Oncology (120 citations), Cancer Research (63 citations), Cell Biology (74 citations) and Infectious Diseases (73 citations). Ruth Eichner has collaborated with scholars based in Germany, Austria and United States. Frequent co-authors include Florian Bassermann, Michele Pagano, Arno Müllbacher, Paul Waring, Giulio Superti‐Furga, Enrico Girardi, Vanesa Fernández‐Sáiz, Bianca‐Sabrina Targosz, Mari Hashimoto and Julia Slotta‐Huspenina. Their work appears in journals such as ACS Chemical Biology, Microbiology, Blood, Nature Cell Biology and Cell Host & Microbe.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.