Ronald E. Gates

821 citations
36 papers · 743 · h-index 16

Impact in

Papers in

    • Protein Kinase Regulation and GTPase Signaling 7
    • Retinoids in leukemia and cellular processes 5
    • Protein Tyrosine Phosphatases 3
    • Glycosylation and Glycoproteins Research 2
    • Skin and Cellular Biology Research 6

Ronald E. Gates

35 papers receiving 680 citations

Peers

Ronald E. Gates
Comparison fields: 5 of 93
  • Immunology and Allergy 109
  • Cell Biology 199
  • Dermatology 59
  • Molecular Biology 449
  • Oncology 160
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Citations per field
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Citations per year

Countries citing papers authored by Ronald E. Gates

Since Specialization
Citations

This map shows the geographic impact of Ronald E. Gates's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ronald E. Gates with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ronald E. Gates more than expected).

Fields of papers citing papers by Ronald E. Gates

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ronald E. Gates. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ronald E. Gates. The network helps show where Ronald E. Gates may publish in the future.

Co-authors

The 22 scholars most cited alongside Ronald E. Gates, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Ronald E. Gates Line = papers co-authored together Ronald E. Gates links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 36 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Potential role for focal adhesion kinase in migrating and proliferating keratinocytes near epidermal wounds and in culture.
199488
2 199084
3 199059
4 198245
5 199143
6 198237
7 198536
8
Altered distribution of phospholipase C-gamma 1 in benign hyperproliferative epidermal diseases.
199235
9 198333
10 197029
11 197826
12 197925
13 199120
14 198718
15 198518
16 199716
17 197112
18 197512
19 198811
20 198810

About Ronald E. Gates

Ronald E. Gates is a scholar working on Molecular Biology, Cell Biology, Immunology and Allergy, Cancer Research and Pathology and Forensic Medicine, having authored 36 papers that have together received 743 indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (7 papers), Skin and Cellular Biology Research (6 papers), Cell Adhesion Molecules Research (6 papers), Retinoids in leukemia and cellular processes (5 papers), Autoimmune Bullous Skin Diseases (4 papers), Protein Tyrosine Phosphatases (3 papers), Mass Spectrometry Techniques and Applications (3 papers) and Glycosylation and Glycoproteins Research (2 papers). The work is most often cited by research in Immunology and Allergy (109 citations), Cell Biology (199 citations), Dermatology (59 citations), Molecular Biology (449 citations) and Oncology (160 citations). Ronald E. Gates has collaborated with scholars based in United States and Australia. Frequent co-authors include Lloyd E. King, Lillian B. Nanney, Christa M. Stoscheck, Steven K. Hanks, Ellen S. Kang, Harvey F. Fisher, Dallas G. Cross, Martin Morrison, G Carpenter and Gordon Todderud. Their work appears in journals such as Biochemical and Biophysical Research Communications, Journal of Investigative Dermatology, Analytical Biochemistry, Biochemical Journal and Journal of Cutaneous Pathology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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