Romana E. Ranftl
Impact in
-
- Colorectal Cancer Treatments and Studies
- Cancer Cells and Metastasis
- Cancer Immunotherapy and Biomarkers
-
- Cancer Genomics and Diagnostics
- Cancer, Hypoxia, and Metabolism
Papers in
-
- Cellular Mechanics and Interactions 3
- Hippo pathway signaling and YAP/TAZ 3
- Oncology 3
- Cancer Cells and Metastasis 2
- Co-authors
- Fernando Calvo (3 shared papers)Emad Moeendarbary (2 shared papers)Aaron J. Farrugia (2 shared papers)Guillaume Charras (1 shared paper)Steven Hooper (2 shared papers)Facundo D. Batista (1 shared paper)Andreas Bruckbauer (1 shared paper)Erik A. Sahai (2 shared papers)
- Journals
- Cancer Cell (1 paper)Nature Communications (1 paper)European Journal of Cancer (1 paper)Cell Reports (1 paper)Methods in molecular biology (1 paper)
- Partner nations
- United KingdomUnited StatesDenmark
In The Last Decade
Romana E. Ranftl
4 papers receiving 464 citations
Peers
Comparison fields: 5 of 58
- Oncology 223
- Cancer Research 112
- Cell Biology 92
- Pathology and Forensic Medicine 57
- Aging 6
Countries citing papers authored by Romana E. Ranftl
This map shows the geographic impact of Romana E. Ranftl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Romana E. Ranftl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Romana E. Ranftl more than expected).
Fields of papers citing papers by Romana E. Ranftl
This network shows the impact of papers produced by Romana E. Ranftl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Romana E. Ranftl. The network helps show where Romana E. Ranftl may publish in the future.
Co-authors
The 16 scholars most cited alongside Romana E. Ranftl, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 219 | |
| 2 | 2019 | 135 | |
| 3 | 2015 | 108 | |
| 4 | 2017 | 7 | |
| 5 | 2016 | 0 |
About Romana E. Ranftl
Romana E. Ranftl is a scholar working on Cell Biology, Oncology, Cancer Research, Pathology and Forensic Medicine and Molecular Biology, having authored 5 papers that have together received 469 indexed citations. Recurring topics across this work include Cellular Mechanics and Interactions (3 papers), Hippo pathway signaling and YAP/TAZ (3 papers), Cancer Cells and Metastasis (2 papers), Cancer Genomics and Diagnostics (1 paper), 3D Printing in Biomedical Research (1 paper), Heat shock proteins research (1 paper), Wnt/β-catenin signaling in development and cancer (1 paper) and Genetic factors in colorectal cancer (1 paper). The work is most often cited by research in Oncology (223 citations), Cancer Research (112 citations), Cell Biology (92 citations), Pathology and Forensic Medicine (57 citations) and Aging (6 citations). Romana E. Ranftl has collaborated with scholars based in United Kingdom, United States and Denmark. Frequent co-authors include Fernando Calvo, Emad Moeendarbary, Aaron J. Farrugia, Guillaume Charras, Steven Hooper, Facundo D. Batista, Andreas Bruckbauer, Erik A. Sahai, Luca Magnani and Maria Semiannikova. Their work appears in journals such as Cancer Cell, Nature Communications, European Journal of Cancer, Cell Reports and Methods in molecular biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.