Robert Saiz

590 citations
9 papers · 508 · h-index 8

Impact in

  • Genetics top 10%
    • Inflammatory Bowel Disease
    • Immunodeficiency and Autoimmune Disorders

Papers in

    • TGF-β signaling in diseases 2
    • Peroxisome Proliferator-Activated Receptors 1
    • CRISPR and Genetic Engineering 1
    • DNA Repair Mechanisms 1

Robert Saiz

9 papers receiving 493 citations

Peers

Robert Saiz
Comparison fields: 5 of 70
  • Genetics 184
  • Immunology 102
  • Oncology 86
  • Molecular Biology 196
  • Gastroenterology 13
Replace M. J. Smith with:
M. J. Smith United Kingdom
Manon Dubé Canada
Cody Fine United States
Iryna Shanina Canada
Hisako Kawata Japan
Helene Will Germany
Ryoichi Iiyama Japan
Saskia Thomas Sweden
Steven Levitte United States
Shannon Chilewski United States
Robert Saiz relative to M. J. Smith United Kingdom M. J. Smith's profile →
Citations per field
00.5×
M. J. Smith · 1×
Citations per year

Countries citing papers authored by Robert Saiz

Since Specialization
Citations

This map shows the geographic impact of Robert Saiz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Saiz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Saiz more than expected).

Fields of papers citing papers by Robert Saiz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Saiz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Saiz. The network helps show where Robert Saiz may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert Saiz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert Saiz Line = papers co-authored together Robert Saiz links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 1999195
2 2001115
3 199977
4 199940
5 200032
6 200621
7 200316
8 20148
9 20214

About Robert Saiz

Robert Saiz is a scholar working on Molecular Biology, Oncology, Genetics, Immunology and Epidemiology, having authored 9 papers that have together received 508 indexed citations. Recurring topics across this work include TGF-β signaling in diseases (2 papers), Inflammatory Bowel Disease (2 papers), Peroxisome Proliferator-Activated Receptors (1 paper), CRISPR and Genetic Engineering (1 paper), Toxoplasma gondii Research Studies (1 paper), Genomic variations and chromosomal abnormalities (1 paper), DNA Repair Mechanisms (1 paper) and Immunodeficiency and Autoimmune Disorders (1 paper). The work is most often cited by research in Genetics (184 citations), Immunology (102 citations), Oncology (86 citations), Molecular Biology (196 citations) and Gastroenterology (13 citations). Robert Saiz has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Phillip A. Morin, Elad Ziv, Jane A. Cauley, W S Browner, Stefan Schreiber, Andrew J. Macpherson, Christopher G. Mathew, Stephen Bridger, Jochen Hampe and Mark Curran. Their work appears in journals such as Journal of Virology, BioTechniques, Immunogenetics, The American Journal of Human Genetics and Clinical & Translational Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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