Richard C. Witt

15 papers receiving 509 citations

Peers

Richard C. Witt
Comparison fields: 5 of 74
  • Pharmacology 170
  • Biochemistry 54
  • Toxicology 20
  • Cancer Research 69
  • Cardiology and Cardiovascular Medicine 68
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Countries citing papers authored by Richard C. Witt

Since Specialization
Citations

This map shows the geographic impact of Richard C. Witt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard C. Witt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard C. Witt more than expected).

Fields of papers citing papers by Richard C. Witt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard C. Witt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard C. Witt. The network helps show where Richard C. Witt may publish in the future.

Co-authors

The 25 scholars most cited alongside Richard C. Witt, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Richard C. Witt Line = papers co-authored together Richard C. Witt links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1
Lipoxygenase inhibitors attenuate growth of human pancreatic cancer xenografts and induce apoptosis through the mitochondrial pathway.
2002137
2 2008129
3
Leukotriene B4 receptor antagonist LY293111 inhibits proliferation and induces apoptosis in human pancreatic cancer cells.
200299
4 199447
5 200430
6 199130
7 199314
8 199410
9 19917
10 20036
11 20036
12 19906
13 19934
14
[Electrophysiological aspects of the effect of creatine phosphate on myocardial cellular activity in the normal state and in ischemia].
19902
15
A Preliminary Catalog of the Lichen-Forming Fungi of Nebraska
19951
16 19921

About Richard C. Witt

Richard C. Witt is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Pharmacology, Cellular and Molecular Neuroscience and Cell Biology, having authored 16 papers that have together received 529 indexed citations. Recurring topics across this work include Cardiac electrophysiology and arrhythmias (3 papers), Ion channel regulation and function (3 papers), Inflammatory mediators and NSAID effects (3 papers), Nerve injury and regeneration (2 papers), Calpain Protease Function and Regulation (2 papers), Signaling Pathways in Disease (2 papers), Botany and Plant Ecology Studies (1 paper) and Lichen and fungal ecology (1 paper). The work is most often cited by research in Pharmacology (170 citations), Biochemistry (54 citations), Toxicology (20 citations), Cancer Research (69 citations) and Cardiology and Cardiovascular Medicine (68 citations). Richard C. Witt has collaborated with scholars based in United States, Russia and United Arab Emirates. Frequent co-authors include Thomas E. Adrian, Weigang Tong, George J. Rozanski, Xianzhong Ding, René Hennig, Jens Standop, Xianzhong Ding, Parviz M. Pour, Xian-Zhong Ding and Mohammad R. Salabat. Their work appears in journals such as American Journal of Physiology-Heart and Circulatory Physiology, Health & Social Work, Clinical Cancer Research, Molecular and Cellular Biochemistry and Circulation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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