Nils Rahner

33 papers receiving 1000 citations

Peers

Nils Rahner
Comparison fields: 5 of 66
  • Pathology and Forensic Medicine 658
  • Cancer Research 331
  • Oncology 445
  • Genetics 230
  • Molecular Biology 310
Replace Clyde Bailey with:
Clyde Bailey United States
Verena Steinke Germany
Barbro Werelius Sweden
Michelle Landon United States
Stéphanie Baert‐Desurmont France
Marjo van Puijenbroek Netherlands
Daniëlle Bodmer Netherlands
Eva Rambech Sweden
JT Wijnen Netherlands
Erin Salo‐Mullen United States
Nils Rahner relative to Clyde Bailey United States Clyde Bailey's profile →
Citations per field
00.5×3.2×
Clyde Bailey · 1×
Citations per year

Countries citing papers authored by Nils Rahner

Since Specialization
Citations

This map shows the geographic impact of Nils Rahner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nils Rahner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nils Rahner more than expected).

Fields of papers citing papers by Nils Rahner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nils Rahner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nils Rahner. The network helps show where Nils Rahner may publish in the future.

Co-authors

The 25 scholars most cited alongside Nils Rahner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Nils Rahner Line = papers co-authored together Nils Rahner links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 35 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2009107
2 2007101
3 200997
4 200879
5 200573
6 200868
7 200754
8 200753
9 201243
10 200938
11 200834
12 201428
13 200626
14 201125
15 201122
16 201021
17 201719
18 201916
19 200715
20 200815

About Nils Rahner

Nils Rahner is a scholar working on Pathology and Forensic Medicine, Molecular Biology, Oncology, Cancer Research and Genetics, having authored 35 papers that have together received 1.0k indexed citations. Recurring topics across this work include Genetic factors in colorectal cancer (25 papers), Cancer Genomics and Diagnostics (13 papers), Colorectal Cancer Screening and Detection (10 papers), RNA Research and Splicing (4 papers), Genomic variations and chromosomal abnormalities (4 papers), DNA Repair Mechanisms (3 papers), Colorectal Cancer Treatments and Studies (2 papers) and Galectins and Cancer Biology (2 papers). The work is most often cited by research in Pathology and Forensic Medicine (658 citations), Cancer Research (331 citations), Oncology (445 citations), Genetics (230 citations) and Molecular Biology (310 citations). Nils Rahner has collaborated with scholars based in Germany, France and United Kingdom. Frequent co-authors include Verena Steinke, Peter Propping, Nicolaus Friedrichs, Stefan Aretz, Waltraut Friedl, Reinhard Buettner, Elisabeth Mangold, Reinhard Büttner, Constanze Walldorf and Frank Lammert. Their work appears in journals such as European Journal of Human Genetics, Human Mutation, Familial Cancer, Human Genetics and Journal of Clinical Oncology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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