Mohammed Tadayyon

14 papers receiving 1.1k citations

Peers

Mohammed Tadayyon
Comparison fields: 5 of 78
  • Endocrine and Autonomic Systems 291
  • Cognitive Neuroscience 275
  • Pediatrics, Perinatology and Child Health 156
  • Cellular and Molecular Neuroscience 92
  • Genetics 147
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Citations per field
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Citations per year

Countries citing papers authored by Mohammed Tadayyon

Since Specialization
Citations

This map shows the geographic impact of Mohammed Tadayyon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mohammed Tadayyon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mohammed Tadayyon more than expected).

Fields of papers citing papers by Mohammed Tadayyon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mohammed Tadayyon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mohammed Tadayyon. The network helps show where Mohammed Tadayyon may publish in the future.

Co-authors

The 25 scholars most cited alongside Mohammed Tadayyon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mohammed Tadayyon Line = papers co-authored together Mohammed Tadayyon links everyone, so they are left out of the graph.

All Works

14 of 14 papers shown
#Work
1 2000282
2 2011279
3 1999266
4 1990116
5 199651
6 199427
7 199218
8 199815
9 199010
10 19888
11
Expression of membrane proteins in Escherichia coli
19946
12 19925
13 19874
14
Pharmacological interference with phospholipase A2 activity reveals mechanistic differences between glucose and glyceraldehyde induced insulin release: implication for coupling of glucose metabolism to phospholipase A2 activity.
19932

About Mohammed Tadayyon

Mohammed Tadayyon is a scholar working on Endocrine and Autonomic Systems, Genetics, Pediatrics, Perinatology and Child Health, Surgery and Epidemiology, having authored 14 papers that have together received 1.1k indexed citations. Recurring topics across this work include Bacterial Genetics and Biotechnology (5 papers), Pancreatic function and diabetes (4 papers), Regulation of Appetite and Obesity (3 papers), Epigenetics and DNA Methylation (1 paper), Neonatal Health and Biochemistry (1 paper), Adipokines, Inflammation, and Metabolic Diseases (1 paper), Genomics and Chromatin Dynamics (1 paper) and Birth, Development, and Health (1 paper). The work is most often cited by research in Endocrine and Autonomic Systems (291 citations), Cognitive Neuroscience (275 citations), Pediatrics, Perinatology and Child Health (156 citations), Cellular and Molecular Neuroscience (92 citations) and Genetics (147 citations). Mohammed Tadayyon has collaborated with scholars based in United Kingdom, United Arab Emirates and Sweden. Frequent co-authors include Jenny K. Broome‐Smith, Jonathan R.S. Arch, Andrea C. Haynes, Helen Chapman, Roderick A. Porter, Amanda Johns, Peter S. Widdowson, Robin E. Buckingham, John P.H. Wilding and Joanne Allison Harrold. Their work appears in journals such as Journal of Endocrinology, Molecular Membrane Biology, Life Sciences, Proceedings of the National Academy of Sciences and Diabetes.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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