Mitchell W. Dillon
Impact in
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- Genomics and Rare Diseases
- Genomic variations and chromosomal abnormalities
- BRCA gene mutations in cancer
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- Galectins and Cancer Biology
Papers in
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- Protein Tyrosine Phosphatases 4
- PI3K/AKT/mTOR signaling in cancer 1
- Co-authors
- Heather Mason‐Suares (4 shared papers)Jennifer A. Lee (2 shared papers)Katherine A. Rauen (2 shared papers)Lisa M. Vincent (2 shared papers)Hélène Cavé (2 shared papers)Bruce D. Gelb (2 shared papers)Karen W. Gripp (2 shared papers)Martin Zenker (2 shared papers)
- Journals
- Genetics in Medicine (1 paper)Prenatal Diagnosis (1 paper)Human Mutation (1 paper)Clinical Pharmacology & Therapeutics (1 paper)Molecular Genetics & Genomic Medicine (2 papers)
- Partner nations
- United StatesGermanyFrance
In The Last Decade
Mitchell W. Dillon
7 papers receiving 206 citations
Peers
Comparison fields: 5 of 34
- Genetics 69
- Immunology 45
- Molecular Biology 110
- Cancer Research 15
- Neurology 13
Countries citing papers authored by Mitchell W. Dillon
This map shows the geographic impact of Mitchell W. Dillon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mitchell W. Dillon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mitchell W. Dillon more than expected).
Fields of papers citing papers by Mitchell W. Dillon
This network shows the impact of papers produced by Mitchell W. Dillon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mitchell W. Dillon. The network helps show where Mitchell W. Dillon may publish in the future.
Co-authors
The 25 scholars most cited alongside Mitchell W. Dillon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 92 | |
| 2 | 2018 | 62 | |
| 3 | 2016 | 25 | |
| 4 | 2019 | 12 | |
| 5 | 2020 | 7 | |
| 6 | 2016 | 5 | |
| 7 | 2020 | 4 |
About Mitchell W. Dillon
Mitchell W. Dillon is a scholar working on Molecular Biology, Organic Chemistry, Pediatrics, Perinatology and Child Health, Pulmonary and Respiratory Medicine and Oncology, having authored 7 papers that have together received 207 indexed citations. Recurring topics across this work include Protein Tyrosine Phosphatases (4 papers), Pharmacogenetics and Drug Metabolism (1 paper), Genomics and Rare Diseases (1 paper), Pharmacological Effects and Toxicity Studies (1 paper), Galectins and Cancer Biology (1 paper), PI3K/AKT/mTOR signaling in cancer (1 paper), Peptidase Inhibition and Analysis (1 paper) and Cystic Fibrosis Research Advances (1 paper). The work is most often cited by research in Genetics (69 citations), Immunology (45 citations), Molecular Biology (110 citations), Cancer Research (15 citations) and Neurology (13 citations). Mitchell W. Dillon has collaborated with scholars based in United States, Germany and France. Frequent co-authors include Heather Mason‐Suares, Jennifer A. Lee, Katherine A. Rauen, Lisa M. Vincent, Hélène Cavé, Bruce D. Gelb, Karen W. Gripp, Martin Zenker, Bradley A. Williams and Matthew S. Lebo. Their work appears in journals such as Genetics in Medicine, Prenatal Diagnosis, Human Mutation, Clinical Pharmacology & Therapeutics and Molecular Genetics & Genomic Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.