Miriam Döcker
Impact in
- Psychiatry and Mental health top 2%
- Epilepsy research and treatment
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- Genomics and Rare Diseases
- Genetics and Neurodevelopmental Disorders
Papers in
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- Epilepsy research and treatment 5
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- Cellular transport and secretion 1
- Co-authors
- Saskia Biskup (2 shared papers)Johannes R. Lemke (2 shared papers)Thomas Bast (1 shared paper)Nunzio Iraci (1 shared paper)KLAUS HARZER (1 shared paper)Antje Bornemann (1 shared paper)Florian Battke (1 shared paper)Ingeborg Krägeloh‐Mann (1 shared paper)
- Journals
- Neurology (1 paper)Annals of Neurology (1 paper)Frontiers in Neurology (1 paper)Acta Neuropathologica (1 paper)Journal of Inherited Metabolic Disease (1 paper)
- Partner nations
- GermanyNetherlandsFrance
In The Last Decade
Miriam Döcker
6 papers receiving 760 citations
Miriam Döcker's Hit Papers
Peers
Comparison fields: 5 of 32
- Psychiatry and Mental health 581
- Genetics 112
- Molecular Biology 172
- Cellular and Molecular Neuroscience 27
- Neurology 21
Countries citing papers authored by Miriam Döcker
This map shows the geographic impact of Miriam Döcker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miriam Döcker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miriam Döcker more than expected).
Fields of papers citing papers by Miriam Döcker
This network shows the impact of papers produced by Miriam Döcker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miriam Döcker. The network helps show where Miriam Döcker may publish in the future.
Co-authors
The 25 scholars most cited alongside Miriam Döcker, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Genetic and phenotypic heterogeneity suggest therapeutic implications in SCN2A-related disorders Hit paper breakdown → | 2017 | 460 |
| 2 | 2015 | 243 | |
| 3 | 2018 | 40 | |
| 4 | 2016 | 26 | |
| 5 | 2016 | 16 | |
| 6 | 2024 | 5 | |
| 7 | 2023 | 0 |
About Miriam Döcker
Miriam Döcker is a scholar working on Psychiatry and Mental health, Cell Biology, Cellular and Molecular Neuroscience, Physiology and Genetics, having authored 7 papers that have together received 790 indexed citations. Recurring topics across this work include Epilepsy research and treatment (5 papers), Cellular transport and secretion (1 paper), Lysosomal Storage Disorders Research (1 paper), Ion channel regulation and function (1 paper), Genetic Neurodegenerative Diseases (1 paper) and Genomics and Rare Diseases (1 paper). The work is most often cited by research in Psychiatry and Mental health (581 citations), Genetics (112 citations), Molecular Biology (172 citations), Cellular and Molecular Neuroscience (27 citations) and Neurology (21 citations). Miriam Döcker has collaborated with scholars based in Germany, Netherlands and France. Frequent co-authors include Saskia Biskup, Johannes R. Lemke, Thomas Bast, Nunzio Iraci, KLAUS HARZER, Antje Bornemann, Florian Battke, Ingeborg Krägeloh‐Mann, Ilaria Mosca and Maurizio Taglialatela. Their work appears in journals such as Neurology, Annals of Neurology, Frontiers in Neurology, Acta Neuropathologica and Journal of Inherited Metabolic Disease.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.