Mike Trus

2k citations
48 papers · 1.8k · h-index 22

Impact in

    • Cellular transport and secretion
    • Ion channel regulation and function
    • Genomics and Chromatin Dynamics
    • Redox biology and oxidative stress

Papers in

    • Cellular transport and secretion 10
    • Ion channel regulation and function 17
    • Genomics and Chromatin Dynamics 5
    • Redox biology and oxidative stress 4
    • Metabolism, Diabetes, and Cancer 3

Mike Trus

47 papers receiving 1.7k citations

Peers

Mike Trus
Comparison fields: 5 of 85
  • Cell Biology 244
  • Molecular Biology 770
  • Biochemistry 64
  • Oncology 201
  • Hematology 113
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Eloise C. Anthony Netherlands
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Citations per year

Countries citing papers authored by Mike Trus

Since Specialization
Citations

This map shows the geographic impact of Mike Trus's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mike Trus with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mike Trus more than expected).

Fields of papers citing papers by Mike Trus

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mike Trus. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mike Trus. The network helps show where Mike Trus may publish in the future.

Co-authors

The 25 scholars most cited alongside Mike Trus, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mike Trus Line = papers co-authored together Mike Trus links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 48 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2012410
2 1999251
3 199788
4 199769
5 201866
6 200557
7 201155
8 199653
9 200153
10 201747
11 199443
12
N-type voltage-sensitive calcium channel interacts with syntaxin, synaptotagmin and SNAP-25 in a multiprotein complex.
199843
13 200142
14 200742
15 201340
16 199039
17 200639
18 200138
19 200137
20 201827

About Mike Trus

Mike Trus is a scholar working on Cell Biology, Molecular Biology, Hematology, Hepatology and Oncology, having authored 48 papers that have together received 1.8k indexed citations. Recurring topics across this work include Ion channel regulation and function (17 papers), Cellular transport and secretion (10 papers), Acute Myeloid Leukemia Research (6 papers), Genomics and Chromatin Dynamics (5 papers), Redox biology and oxidative stress (4 papers), Metabolism, Diabetes, and Cancer (3 papers), Cancer Cells and Metastasis (2 papers) and Asthma and respiratory diseases (2 papers). The work is most often cited by research in Cell Biology (244 citations), Molecular Biology (770 citations), Biochemistry (64 citations), Oncology (201 citations) and Hematology (113 citations). Mike Trus has collaborated with scholars based in Israel, Canada and United States. Frequent co-authors include Daphné Atlas, Ofer Wiser, Dror Tobi, Erik Renström, Sebastian Barg, Patrik Rorsman, Lea Reshef, Nissim Benvenisty, Mark David Minden and Louise J. Bordeleau. Their work appears in journals such as Scientific Reports, Blood, FEBS Letters, Leukemia and Cell Calcium.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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