Mayuko Inuzuka

459 citations
11 papers · 249 · h-index 6

Impact in

Papers in

    • BRCA gene mutations in cancer 10
    • Genomic variations and chromosomal abnormalities 4
    • Nutrition, Genetics, and Disease 3
    • Genetic Associations and Epidemiology 1
    • DNA Repair Mechanisms 3

Mayuko Inuzuka

9 papers receiving 241 citations

Peers

Mayuko Inuzuka
Comparison fields: 5 of 37
  • Cancer Research 58
  • Pathology and Forensic Medicine 58
  • Genetics 87
  • Oncology 76
  • Reproductive Medicine 22
Replace René Scheiden with:
René Scheiden Luxembourg
Benjamin Goode United States
Petra Zemánková Czechia
Giona Turri Italy
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Asad Haider United States
Hy-Sook Kim South Korea
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Citations per field
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Citations per year

Countries citing papers authored by Mayuko Inuzuka

Since Specialization
Citations

This map shows the geographic impact of Mayuko Inuzuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mayuko Inuzuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mayuko Inuzuka more than expected).

Fields of papers citing papers by Mayuko Inuzuka

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mayuko Inuzuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mayuko Inuzuka. The network helps show where Mayuko Inuzuka may publish in the future.

Co-authors

The 25 scholars most cited alongside Mayuko Inuzuka, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mayuko Inuzuka Line = papers co-authored together Mayuko Inuzuka links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1
Presence of ras oncogene mutations and human papillomavirus DNA in human prostate carcinomas.
1992141
2 200939
3 201918
4 201916
5 202016
6 201915
7 20182
8 20201
9 20151
10 20240
11 20200

About Mayuko Inuzuka

Mayuko Inuzuka is a scholar working on Genetics, Molecular Biology, Pathology and Forensic Medicine, Oncology and Cancer Research, having authored 11 papers that have together received 249 indexed citations. Recurring topics across this work include BRCA gene mutations in cancer (10 papers), Genomic variations and chromosomal abnormalities (4 papers), DNA Repair Mechanisms (3 papers), Nutrition, Genetics, and Disease (3 papers), Cancer Genomics and Diagnostics (2 papers), Genetic factors in colorectal cancer (2 papers), Genetic Associations and Epidemiology (1 paper) and PARP inhibition in cancer therapy (1 paper). The work is most often cited by research in Cancer Research (58 citations), Pathology and Forensic Medicine (58 citations), Genetics (87 citations), Oncology (76 citations) and Reproductive Medicine (22 citations). Mayuko Inuzuka has collaborated with scholars based in Japan, China and United States. Frequent co-authors include Hironobu Naiki, K Nakakuki, Khurshid Anwar, Takuya Shiraishi, Seigo Nakamura, Yoshimi Ide, Junko Yotsumoto, Masami Arai, Reiko Yoshida and Chie Watanabe. Their work appears in journals such as Scientific Reports, Journal of Clinical Oncology, Clinical Breast Cancer, Breast Cancer and Breast Cancer Research and Treatment.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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