Max E. Douglas
Impact in
- Cell Biology top 5%
- Microtubule and mitosis dynamics
- Aging top 10%
Papers in
-
- DNA Repair Mechanisms 9
- CRISPR and Genetic Engineering 3
- Protist diversity and phylogeny 2
- Photosynthetic Processes and Mechanisms 2
-
- Telomeres, Telomerase, and Senescence 4
- Co-authors
- John F.X. Diffley (6 shared papers)Alessandro Costa (4 shared papers)Ferdos Abid Ali (3 shared papers)Masanori Mishima (3 shared papers)Tim Davies (1 shared paper)Nimesh Joseph (1 shared paper)Julia Locke (2 shared papers)Andrea Nans (2 shared papers)
- Journals
- Nature Communications (5 papers)Nature (2 papers)Nucleic Acids Research (2 papers)Current Biology (1 paper)Journal of Biological Chemistry (1 paper)
- Partner nations
- United KingdomUnited StatesSweden
In The Last Decade
Max E. Douglas
14 papers receiving 716 citations
Peers
Comparison fields: 5 of 61
- Cell Biology 250
- Aging 20
- Molecular Biology 623
- Genetics 108
- Structural Biology 4
Countries citing papers authored by Max E. Douglas
This map shows the geographic impact of Max E. Douglas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Max E. Douglas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Max E. Douglas more than expected).
Fields of papers citing papers by Max E. Douglas
This network shows the impact of papers produced by Max E. Douglas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Max E. Douglas. The network helps show where Max E. Douglas may publish in the future.
Co-authors
The 25 scholars most cited alongside Max E. Douglas, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 191 | |
| 2 | 2010 | 111 | |
| 3 | 2018 | 89 | |
| 4 | 2017 | 71 | |
| 5 | 2019 | 64 | |
| 6 | 2017 | 64 | |
| 7 | 2015 | 49 | |
| 8 | 2010 | 46 | |
| 9 | 2021 | 15 | |
| 10 | 2024 | 11 | |
| 11 | 2025 | 3 | |
| 12 | 2025 | 1 | |
| 13 | 2001 | 1 | |
| 14 | 2024 | 1 |
About Max E. Douglas
Max E. Douglas is a scholar working on Molecular Biology, Physiology, Cell Biology, Genetics and Materials Chemistry, having authored 14 papers that have together received 717 indexed citations. Recurring topics across this work include DNA Repair Mechanisms (9 papers), Telomeres, Telomerase, and Senescence (4 papers), Microtubule and mitosis dynamics (3 papers), CRISPR and Genetic Engineering (3 papers), Enzyme Structure and Function (2 papers), Protist diversity and phylogeny (2 papers), Photosynthetic Processes and Mechanisms (2 papers) and Bacterial Genetics and Biotechnology (2 papers). The work is most often cited by research in Cell Biology (250 citations), Aging (20 citations), Molecular Biology (623 citations), Genetics (108 citations) and Structural Biology (4 citations). Max E. Douglas has collaborated with scholars based in United Kingdom, United States and Sweden. Frequent co-authors include John F.X. Diffley, Alessandro Costa, Ferdos Abid Ali, Masanori Mishima, Tim Davies, Nimesh Joseph, Julia Locke, Andrea Nans, Valerie E. Pye and Patrik Eickhoff. Their work appears in journals such as Nature Communications, Nature, Nucleic Acids Research, Current Biology and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.