Matthew E. Jones
Impact in
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- Cancer Genomics and Diagnostics
- Cancer-related molecular mechanisms research
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- Single-cell and spatial transcriptomics
- Extracellular vesicles in disease
- CRISPR and Genetic Engineering
Papers in
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- Ion channel regulation and function 2
- CRISPR and Genetic Engineering 1
- RNA modifications and cancer 1
- RNA regulation and disease 1
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- Cell Adhesion Molecules Research 1
- Co-authors
- Alistair R. R. Forrest (3 shared papers)Timo Lassmann (2 shared papers)Елена Дмитриевна Денисенко (2 shared papers)Belinda B. Guo (1 shared paper)Olivier Clément (1 shared paper)Rui Hou (1 shared paper)Daniel Poppe (1 shared paper)Leanne de Kock (1 shared paper)
In The Last Decade
Matthew E. Jones
8 papers receiving 490 citations
Matthew E. Jones's Hit Papers
Peers
Comparison fields: 5 of 73
- Cancer Research 78
- Molecular Biology 343
- Immunology 95
- Biophysics 24
- Neurology 27
Countries citing papers authored by Matthew E. Jones
This map shows the geographic impact of Matthew E. Jones's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew E. Jones with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew E. Jones more than expected).
Fields of papers citing papers by Matthew E. Jones
This network shows the impact of papers produced by Matthew E. Jones. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew E. Jones. The network helps show where Matthew E. Jones may publish in the future.
Co-authors
The 25 scholars most cited alongside Matthew E. Jones, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Systematic assessment of tissue dissociation and storage biases in single-cell and single-nucleus RNA-seq workflows Hit paper breakdown → | 2020 | 417 |
| 2 | 2023 | 23 | |
| 3 | 2019 | 19 | |
| 4 | 2023 | 15 | |
| 5 | 2022 | 9 | |
| 6 | 2025 | 6 | |
| 7 | 2007 | 2 | |
| 8 | 2025 | 1 |
About Matthew E. Jones
Matthew E. Jones is a scholar working on Molecular Biology, Immunology and Allergy, Microbiology, Animal Science and Zoology and Cancer Research, having authored 8 papers that have together received 492 indexed citations. Recurring topics across this work include Ion channel regulation and function (2 papers), CRISPR and Genetic Engineering (1 paper), Mast cells and histamine (1 paper), RNA modifications and cancer (1 paper), Livestock and Poultry Management (1 paper), Cell Adhesion Molecules Research (1 paper), RNA regulation and disease (1 paper) and Virus-based gene therapy research (1 paper). The work is most often cited by research in Cancer Research (78 citations), Molecular Biology (343 citations), Immunology (95 citations), Biophysics (24 citations) and Neurology (27 citations). Matthew E. Jones has collaborated with scholars based in Australia, Poland and France. Frequent co-authors include Alistair R. R. Forrest, Timo Lassmann, Елена Дмитриевна Денисенко, Belinda B. Guo, Olivier Clément, Rui Hou, Daniel Poppe, Leanne de Kock, Rebecca K. Simmons and Ryan Lister. Their work appears in journals such as Neuro-Oncology, Gene, Veterinary Record, Cell Genomics and Molecular Therapy — Methods & Clinical Development.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.