Markus Hellmuth
Impact in
- Pathology and Forensic Medicine top 10%
- Spine and Intervertebral Disc Pathology
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- Osteoarthritis Treatment and Mechanisms
Papers in
-
- Heme Oxygenase-1 and Carbon Monoxide 2
- Angiogenesis and VEGF in Cancer 1
- Oncology 3
- Chemokine receptors and signaling 3
- Co-authors
- Sven K. Tschoeke (4 shared papers)Arwed Hostmann (4 shared papers)Andreas Oberholzer (3 shared papers)Wolfgang Ertel (3 shared papers)Christoph‐E. Heyde (2 shared papers)Josef Pfeilschifter (4 shared papers)Heiko Mühl (4 shared papers)Wolfgang Ertel (1 shared paper)
- Journals
- Cytokine (1 paper)Journal of Orthopaedic Research® (1 paper)Free Radical Biology and Medicine (1 paper)Carcinogenesis (1 paper)BMC Clinical Pathology (1 paper)
- Partner nations
- GermanyUnited States
In The Last Decade
Markus Hellmuth
8 papers receiving 350 citations
Peers
Comparison fields: 5 of 69
- Pathology and Forensic Medicine 107
- Rheumatology 55
- Pharmacology 56
- Emergency Medicine 16
- Immunology 38
Countries citing papers authored by Markus Hellmuth
This map shows the geographic impact of Markus Hellmuth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Markus Hellmuth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Markus Hellmuth more than expected).
Fields of papers citing papers by Markus Hellmuth
This network shows the impact of papers produced by Markus Hellmuth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Markus Hellmuth. The network helps show where Markus Hellmuth may publish in the future.
Co-authors
The 25 scholars most cited alongside Markus Hellmuth, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 76 | |
| 2 | 2008 | 68 | |
| 3 | 2006 | 58 | |
| 4 | 2007 | 58 | |
| 5 | 2002 | 33 | |
| 6 | 2000 | 29 | |
| 7 | 2004 | 18 | |
| 8 | 2004 | 18 |
About Markus Hellmuth
Markus Hellmuth is a scholar working on Molecular Biology, Oncology, Surgery, Pathology and Forensic Medicine and Biomedical Engineering, having authored 8 papers that have together received 358 indexed citations. Recurring topics across this work include Chemokine receptors and signaling (3 papers), Medical Imaging and Analysis (2 papers), Spinal Fractures and Fixation Techniques (2 papers), Spine and Intervertebral Disc Pathology (2 papers), Heme Oxygenase-1 and Carbon Monoxide (2 papers), Nitric Oxide and Endothelin Effects (1 paper), Eicosanoids and Hypertension Pharmacology (1 paper) and Angiogenesis and VEGF in Cancer (1 paper). The work is most often cited by research in Pathology and Forensic Medicine (107 citations), Rheumatology (55 citations), Pharmacology (56 citations), Emergency Medicine (16 citations) and Immunology (38 citations). Markus Hellmuth has collaborated with scholars based in Germany and United States. Frequent co-authors include Sven K. Tschoeke, Arwed Hostmann, Andreas Oberholzer, Wolfgang Ertel, Christoph‐E. Heyde, Josef Pfeilschifter, Heiko Mühl, Wolfgang Ertel, Yohan Robinson and Jens Paulukat. Their work appears in journals such as Cytokine, Journal of Orthopaedic Research®, Free Radical Biology and Medicine, Carcinogenesis and BMC Clinical Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.