Luming He

408 citations
6 papers · 359 · h-index 4

Impact in

    • Neuroinflammation and Neurodegeneration Mechanisms
    • Sphingolipid Metabolism and Signaling
    • RNA Research and Splicing
    • Lipid Membrane Structure and Behavior
    • RNA and protein synthesis mechanisms
    • RNA modifications and cancer
    • Developmental Biology and Gene Regulation

Papers in

Luming He

5 papers receiving 356 citations

Peers

Luming He
Comparison fields: 5 of 69
  • Neurology 41
  • Molecular Biology 245
  • Biochemistry 24
  • Developmental Neuroscience 12
  • Physiology 72
Replace En Huang with:
En Huang Canada
Emmanuelle C. Genin France
Claus Rodemer Germany
Amanjot Kaur Riar United States
Dong Gil Lee South Korea
Markus Uhrig Germany
Yung-Feng Liao Taiwan
Melanie Loix Belgium
Illari Salvatori Italy
Dmitry Grachev Russia
Luming He relative to En Huang Canada En Huang's profile →
Citations per field
00.5×1.5×
En Huang · 1×
Citations per year

Countries citing papers authored by Luming He

Since Specialization
Citations

This map shows the geographic impact of Luming He's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Luming He with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Luming He more than expected).

Fields of papers citing papers by Luming He

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Luming He. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Luming He. The network helps show where Luming He may publish in the future.

Co-authors

The 15 scholars most cited alongside Luming He, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Luming He Line = papers co-authored together Luming He links everyone, so they are left out of the graph.

All Works

About Luming He

Luming He is a scholar working on Molecular Biology, Pathology and Forensic Medicine, Pulmonary and Respiratory Medicine, Geochemistry and Petrology and Neurology, having authored 6 papers that have together received 359 indexed citations. Recurring topics across this work include Zebrafish Biomedical Research Applications (1 paper), Nitric Oxide and Endothelin Effects (1 paper), Sphingolipid Metabolism and Signaling (1 paper), MicroRNA in disease regulation (1 paper), Craniofacial Disorders and Treatments (1 paper), Hedgehog Signaling Pathway Studies (1 paper), Geological Modeling and Analysis (1 paper) and Neuroinflammation and Neurodegeneration Mechanisms (1 paper). The work is most often cited by research in Neurology (41 citations), Molecular Biology (245 citations), Biochemistry (24 citations), Developmental Neuroscience (12 citations) and Physiology (72 citations). Luming He has collaborated with scholars based in United States, China and Pakistan. Frequent co-authors include Jan Xu, Chung Y. Hsu, Chen-Hsiung Yeh, Shawei Chen, Dennis W. Choi, Stefano L. Sensi, Lorella M.T. Canzoniero, Chiara Gamberi, David S. Peterson and Ellen Gottlieb. Their work appears in journals such as Biochemical and Biophysical Research Communications, Stroke, Development, Cancer Research and Journal of Biological Chemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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