Laura Spahn
Impact in
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- Cancer-related molecular mechanisms research
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- Epigenetics and DNA Methylation
- Genomics and Chromatin Dynamics
- RNA modifications and cancer
- RNA Research and Splicing
- Cancer-related gene regulation
Papers in
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- Epigenetics and DNA Methylation 3
- Genomics and Chromatin Dynamics 2
- Cancer-related gene regulation 2
- RNA Research and Splicing 2
- DNA Repair Mechanisms 1
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- Sarcoma Diagnosis and Treatment 4
- Co-authors
- Thomas Jenuwein (1 shared paper)Aditya K. Sengupta (1 shared paper)Stefan Kubicek (1 shared paper)Karl Mechtler (1 shared paper)Anton Wutz (1 shared paper)Stefan Schoeftner (1 shared paper)Haruhiko Koseki (1 shared paper)Heinrich Kovar (4 shared papers)
In The Last Decade
Laura Spahn
7 papers receiving 557 citations
Peers
Comparison fields: 5 of 49
- Cancer Research 99
- Molecular Biology 485
- Genetics 128
- Pulmonary and Respiratory Medicine 116
- Oncology 55
Countries citing papers authored by Laura Spahn
This map shows the geographic impact of Laura Spahn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laura Spahn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laura Spahn more than expected).
Fields of papers citing papers by Laura Spahn
This network shows the impact of papers produced by Laura Spahn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laura Spahn. The network helps show where Laura Spahn may publish in the future.
Co-authors
The 25 scholars most cited alongside Laura Spahn, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 316 | |
| 2 | 2005 | 66 | |
| 3 | 2004 | 56 | |
| 4 | Interaction of the EWS NH2 terminus with BARD1 links the Ewing's sarcoma gene to a common tumor suppressor pathway. | 2002 | 44 |
| 5 | 2003 | 43 | |
| 6 | The EWS protein is dispensable for Ewing tumor growth. | 2001 | 22 |
| 7 | 2006 | 16 |
About Laura Spahn
Laura Spahn is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine, Genetics, Pathology and Forensic Medicine and Pediatrics, Perinatology and Child Health, having authored 7 papers that have together received 563 indexed citations. Recurring topics across this work include Sarcoma Diagnosis and Treatment (4 papers), Epigenetics and DNA Methylation (3 papers), Genomics and Chromatin Dynamics (2 papers), Lymphoma Diagnosis and Treatment (2 papers), Cancer-related gene regulation (2 papers), RNA Research and Splicing (2 papers), Genetics and Neurodevelopmental Disorders (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Cancer Research (99 citations), Molecular Biology (485 citations), Genetics (128 citations), Pulmonary and Respiratory Medicine (116 citations) and Oncology (55 citations). Laura Spahn has collaborated with scholars based in Austria, Germany and Japan. Frequent co-authors include Thomas Jenuwein, Aditya K. Sengupta, Stefan Kubicek, Karl Mechtler, Anton Wutz, Stefan Schoeftner, Haruhiko Koseki, Heinrich Kovar, Christine Siligan and Dave N.T. Aryee. Their work appears in journals such as Oncogene, Cytogenetic and Genome Research, The EMBO Journal, Cancer Research and PubMed.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.