Kinji Ohno

18.0k citations

345 papers · 12.6k · 3 hit papers · h-index 62

Impact in

Neurology top 0.1%
- Myasthenia Gravis and Thymoma
Developmental Neuroscience top 0.5%

Papers in

Molecular Biology 191
- Ion channel regulation and function 49
- RNA Research and Splicing 40
- RNA and protein synthesis mechanisms 29
- Mitochondrial Function and Pathology 26
- RNA modifications and cancer 22
Neurology 96
- Myasthenia Gravis and Thymoma 68

Co-authors: Andrew G. Engel (66 shared papers)Mikako Ito (97 shared papers)Akio Masuda (76 shared papers)Steven M. Sine (28 shared papers)Masaaki Hirayama (34 shared papers)Joan M. Brengman (24 shared papers)Margherita Milone (23 shared papers)Bisei Ohkawara (55 shared papers)
Journals: Scientific Reports (21 papers)Biochemical and Biophysical Research Communications (15 papers)PLoS ONE (12 papers)Neurology (12 papers)International Journal of Molecular Sciences (9 papers)
Partner nations: Japan United States Türkiye

In The Last Decade

Kinji Ohno

333 papers receiving 12.4k citations

Kinji Ohno's Hit Papers

align trajectories log scale

Fusobacterium infection facilitates the development of endometriosis through the phenotypic transition of endometrial fibroblasts 2023 · 97 citations

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if any of the following hold:

it has ≥500 total citations;
it reaches ≥1.5× the top-1% citation threshold for papers in the same subfield and year (the threshold is the minimum needed to enter the top 1%, not the average within it);
it reaches the top citation threshold in at least one of its specific research topics.

2023 Science Translational Medicine
2020 Movement Disorders
2015 PLoS ONE

Peers

Countries citing papers authored by Kinji Ohno

Since Specialization

Citations

This map shows the geographic impact of Kinji Ohno's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kinji Ohno with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kinji Ohno more than expected).

Fields of papers citing papers by Kinji Ohno

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kinji Ohno. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kinji Ohno. The network helps show where Kinji Ohno may publish in the future.

Co-authors

The 25 scholars most cited alongside Kinji Ohno, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Kinji Ohno Line = papers co-authored together Kinji Ohno links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 345 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Intestinal Dysbiosis and Lowered Serum Lipopolysaccharide-Binding Protein in Parkinson’s Disease PLoS ONE ·Satoru Hasegawa, Sae Goto, Hirokazu Tsuji, Tatsuya Okuno, Takashi Asahara, Koji Nomoto, Akihide Shibata, Yoshiro Fujisawa, Tomomi Minato, Akira Okamoto, Kinji Ohno, Masaaki Hirayama Hit paper breakdown →	2015	387
2	Increase of deleted mitochondrial DNA in the striatum in Parkinson's disease and senescence Biochemical and Biophysical Research Communications ·Shin-ichiro Ikebe, Masashi Tanaka, Kinji Ohno, Wataru Sato, Kazuki Hattori, Tomoyoshi Kondo, Yoshikuni Mizuno, Takayuki Ozawa	1990	277
3	Meta‐Analysis of Gut Dysbiosis in Parkinson's Disease Movement Disorders ·Hiroshi Nishiwaki, Mikako Ito, Tomohiro Ishida, Tomonari Hamaguchi, Tetsuya Maeda, Kenichi Kashihara, Yoshio Tsuboi, Jun Ueyama, Teppei Shimamura, Hiroshi Mori, Ken Kurokawa, Masahisa Katsuno, Masaaki Hirayama, Kinji Ohno Hit paper breakdown →	2020	270
4	Mutation of the acetylcholine receptor α subunit causes a slow-channel myasthenic syndrome by enhancing agonist binding affinity Neuron ·Steven M. Sine, Kinji Ohno, Cecilia Bouzat, Anthony Auerbach, Margherita Milone, J. Ned Pruitt, Andrew G. Engel	1995	214
5	Beneficial biological effects and the underlying mechanisms of molecular hydrogen - comprehensive review of 321 original articles - Medical Gas Research ·Masatoshi Ichihara, Sayaka Sobue, Mikako Ito, Masafumi Ito, Masaaki Hirayama, Kinji Ohno	2015	214
6	Progression of Parkinson's disease is associated with gut dysbiosis: Two-year follow-up study PLoS ONE ·Tomomi Minato, Tetsuya Maeda, Yoshiro Fujisawa, Hirokazu Tsuji, Koji Nomoto, Kinji Ohno, Masaaki Hirayama	2017	208
7	Congenital myasthenic syndrome caused by prolonged acetylcholine receptor channel openings due to a mutation in the M2 domain of the epsilon subunit. Proceedings of the National Academy of Sciences ·Kinji Ohno, David Hutchinson, Margherita Milone, Joan M. Brengman, Cecilia Bouzat, Steven M. Sine, Andrew G. Engel	1995	208
8	Human endplate acetylcholinesterase deficiency caused by mutations in the collagen-like tail subunit (ColQ) of the asymmetric enzyme Proceedings of the National Academy of Sciences ·Kinji Ohno, Joan M. Brengman, Akira Tsujino, Andrew G. Engel	1998	199
9	Choline acetyltransferase mutations cause myasthenic syndrome associated with episodic apnea in humans Proceedings of the National Academy of Sciences ·Kinji Ohno, Akira Tsujino, Joan M. Brengman, C. Michel Harper, Željko Bajzer, Bjarne Udd, Roger Beyring, S. Robb, Fenella J. Kirkham, Andrew G. Engel	2001	196
10	Human branch point consensus sequence is yUnAy Nucleic Acids Research ·Kaiping Gao, Akio Masuda, Tohru Matsuura, Kinji Ohno	2008	190
11	Congenital Myasthenic Syndrome Caused by Decreased Agonist Binding Affinity Due to a Mutation in the Acetylcholine Receptor ε Subunit Neuron ·Kinji Ohno, Hai-Long Wang, Margherita Milone, Nina Bren, Joan M. Brengman, Satoshi Nakano, Polly A. Quiram, J. Ned Pruitt, Steven M. Sine, Andrew G. Engel	1996	178
12	Molecular hydrogen is protective against 6-hydroxydopamine-induced nigrostriatal degeneration in a rat model of Parkinson's disease Neuroscience Letters ·Yuan Fu, Mikako Ito, Yasunori Fujita, Masafumi Ito, Masatoshi Ichihara, Akio Masuda, Yumi Suzuki, Satoshi Maesawa, Yasukazu Kajita, Masaaki Hirayama, Ikuroh Ohsawa, Shigeo Ohta, Kinji Ohno	2009	176
13	Position-dependent FUS-RNA interactions regulate alternative splicing events and transcriptions Scientific Reports ·Shinsuke Ishigaki, Akio Masuda, Yusuke Fujioka, Yohei Iguchi, Masahisa Katsuno, Akihide Shibata, Fumihiko Urano, Gen Sobue, Kinji Ohno	2012	173
14	Rapsyn Mutations in Humans Cause Endplate Acetylcholine-Receptor Deficiency and Myasthenic Syndrome The American Journal of Human Genetics ·Kinji Ohno, Andrew G. Engel, Xin‐Ming Shen, Duygu Selcen, Joan M. Brengman, C. Michel Harper, Akira Tsujino, Margherita Milone	2002	170
15	Niemann-Pick C1 Disease: Correlations between NPC1 Mutations, Levels of NPC1 Protein, and Phenotypes Emphasize the Functional Significance of the Putative Sterol-Sensing Domain and of the Cysteine-Rich Luminal Loop The American Journal of Human Genetics ·Gilles Millat, Christophe Marçais, Catherine Tomasetto, Karim Chikh, Anthony H. Fensom, K. Harzer, David A. Wenger, Kinji Ohno, Marie T. Vanier	2001	157
16	Quantitative determination of deleted mitochondrial DNA relative to normal DNA in parkinsonian striatum by a kinetic PCR analysis Biochemical and Biophysical Research Communications ·Takayuki Ozawa, Masashi Tanaka, Shin-ichiro Ikebe, Kinji Ohno, Tomoyoshi Kondo, Yoshikuni Mizuno	1990	153
17	CUGBP1 and MBNL1 preferentially bind to 3′ UTRs and facilitate mRNA decay Scientific Reports ·Akio Masuda, Henriette S. Andersen, Thomas Koed Doktor, Takaaki Okamoto, Mikako Ito, Brage Storstein Andresen, Kinji Ohno	2012	145
18	Molecular Hydrogen as an Emerging Therapeutic Medical Gas for Neurodegenerative and Other Diseases Oxidative Medicine and Cellular Longevity ·Kinji Ohno, Mikako Ito, Masatoshi Ichihara, Masafumi Ito	2012	141
19	Multiple mitochondrial DNA deletions exist in cardiomyocytes of patients with hypertrophic or dilated cardiomyopathy Biochemical and Biophysical Research Communications ·Takayuki Ozawa, Masashi Tanaka, Satoru Sugiyama, Kazuki Hattori, Takayuki Ito, Kinji Ohno, Akira Takahashi, Wataru Sato, Goro Takada, Bunji Mayumi, Kiichirô Yamamoto, Kyo Adachi, Yoshinori Koga, Hironori Toshima	1990	140
20	Myasthenic syndrome caused by mutation of the SCN4A sodium channel Proceedings of the National Academy of Sciences ·Akira Tsujino, Chantal Maertens, Kinji Ohno, Xin‐Ming Shen, Taku Fukuda, Charles M. Harper, Stephen C. Cannon, Andrew G. Engel	2003	140

About Kinji Ohno

Kinji Ohno is a scholar working on Molecular Biology, Neurology, Cell Biology, Physiology and Surgery, having authored 345 papers that have together received 12.6k indexed citations. Recurring topics across this work include Myasthenia Gravis and Thymoma (68 papers), Ion channel regulation and function (49 papers), RNA Research and Splicing (40 papers), Cellular transport and secretion (35 papers), RNA and protein synthesis mechanisms (29 papers), Mitochondrial Function and Pathology (26 papers), Hydrogen's biological and therapeutic effects (25 papers) and RNA modifications and cancer (22 papers). The work is most often cited by research in Neurology (3.8k citations), Developmental Neuroscience (553 citations), Clinical Biochemistry (762 citations), Molecular Biology (7.5k citations) and Cell Biology (1.7k citations). Kinji Ohno has collaborated with scholars based in Japan, United States and Türkiye. Frequent co-authors include Andrew G. Engel, Mikako Ito, Akio Masuda, Steven M. Sine, Masaaki Hirayama, Joan M. Brengman, Margherita Milone, Bisei Ohkawara, Akira Tsujino and Xin‐Ming Shen. Their work appears in journals such as Scientific Reports, Biochemical and Biophysical Research Communications, PLoS ONE, Neurology and International Journal of Molecular Sciences.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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Kinji Ohno

Kinji Ohno's Hit Papers

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About Kinji Ohno

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