John Tat

442 citations
17 papers · 323 · h-index 6

Impact in

    • Cellular transport and secretion
    • Endoplasmic Reticulum Stress and Disease
    • Microtubule and mitosis dynamics
    • Calcium signaling and nucleotide metabolism

Papers in

John Tat

17 papers receiving 319 citations

Peers

John Tat
Comparison fields: 5 of 77
  • Cell Biology 105
  • Physiology 15
  • Molecular Biology 187
  • Oncology 43
  • Cancer Research 17
Replace Samuel J. Rodgers with:
Samuel J. Rodgers Australia
Maria Fernandes Canada
Lauren A. Howell United States
Fumihiro Ishikawa Japan
Annie Lauzier Canada
Fabien Zassadowski France
Dominic B. Bernkopf Germany
Isabelle Hamer Belgium
Pablo Luján Spain
Ayman Mahdy United States
John Tat relative to Samuel J. Rodgers Australia Samuel J. Rodgers's profile →
Citations per field
00.5×5.2×
Samuel J. Rodgers · 1×
Citations per year

Countries citing papers authored by John Tat

Since Specialization
Citations

This map shows the geographic impact of John Tat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Tat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Tat more than expected).

Fields of papers citing papers by John Tat

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John Tat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Tat. The network helps show where John Tat may publish in the future.

Co-authors

The 25 scholars most cited alongside John Tat, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John Tat Line = papers co-authored together John Tat links everyone, so they are left out of the graph.

All Works

17 of 17 papers shown
#Work
1 2014196
2 201336
3 202131
4 201414
5 20229
6 20247
7 20175
8 20145
9 20224
10 20234
11 20203
12 20152
13 20232
14
Strategy for Sustaining Cancer Education Services for Underserved Communities.
20172
15 20261
16 20131
17 20171

About John Tat

John Tat is a scholar working on Molecular Biology, Oncology, General Health Professions, Rheumatology and Cell Biology, having authored 17 papers that have together received 323 indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (4 papers), DNA Repair Mechanisms (3 papers), Folate and B Vitamins Research (2 papers), Microtubule and mitosis dynamics (2 papers), Vitamin C and Antioxidants Research (1 paper), Health Policy Implementation Science (1 paper), Aortic Disease and Treatment Approaches (1 paper) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in Cell Biology (105 citations), Physiology (15 citations), Molecular Biology (187 citations), Oncology (43 citations) and Cancer Research (17 citations). John Tat has collaborated with scholars based in United States, Egypt and United Kingdom. Frequent co-authors include David M. Cowan, Juliati Rahajeng, Suzette Farber-Katz, Michelle M. Ng, Huilin Zhou, Matthew D. Buschman, Christopher Noakes, Marshall C. Peterman, Seth J. Field and Gerry R. Boss. Their work appears in journals such as Clinical Toxicology, Toxics, JACC Basic to Translational Science, Oncogene and Molecular Cell.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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