John Bell

429 citations
21 papers · 319 · h-index 13

Impact in

Papers in

    • Immune Cell Function and Interaction 5
    • Immune Response and Inflammation 3
    • Asthma and respiratory diseases 3

John Bell

19 papers receiving 292 citations

Peers

John Bell
Comparison fields: 5 of 70
  • Physiology 17
  • Health, Toxicology and Mutagenesis 47
  • Immunology 59
  • Physiology 65
  • Pulmonary and Respiratory Medicine 70
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Citations per field
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Citations per year

Countries citing papers authored by John Bell

Since Specialization
Citations

This map shows the geographic impact of John Bell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Bell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Bell more than expected).

Fields of papers citing papers by John Bell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John Bell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Bell. The network helps show where John Bell may publish in the future.

Co-authors

The 25 scholars most cited alongside John Bell, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John Bell Line = papers co-authored together John Bell links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 21 papers — load more, or switch the sort, to bring in the rest.

#Work
1 202433
2 200133
3 202229
4 202225
5 202325
6 201725
7 201225
8
Immunosuppressive and antitumor activity of periodate oxidation product of beta-D-ribosyl-6-methylthiopurine.
196820
9 201619
10 201217
11 202216
12
Inhibition of thymidylate kinase and DNA polymerase by the periodate oxidation product of beta-D-ribosyl-6-methylthiopurine.
196814
13 196914
14 20178
15 19696
16 19695
17 19693
18 20251
19 19691
20 20250

About John Bell

John Bell is a scholar working on Immunology, Physiology, Emergency Medical Services, Oncology and Pulmonary and Respiratory Medicine, having authored 21 papers that have together received 319 indexed citations. Recurring topics across this work include Immune Cell Function and Interaction (5 papers), Polyomavirus and related diseases (4 papers), Cytomegalovirus and herpesvirus research (3 papers), Biochemical and Molecular Research (3 papers), Asthma and respiratory diseases (3 papers), Immune Response and Inflammation (3 papers), Inhalation and Respiratory Drug Delivery (2 papers) and Pediatric health and respiratory diseases (2 papers). The work is most often cited by research in Physiology (17 citations), Health, Toxicology and Mutagenesis (47 citations), Immunology (59 citations), Physiology (65 citations) and Pulmonary and Respiratory Medicine (70 citations). John Bell has collaborated with scholars based in United Kingdom, United States and Sweden. Frequent co-authors include Ekaterina Maslova, Gerald A. LePage, Roland H. Gisler, Aubrey P. Kimball, Alex Wilkinson, Christer Janson, Derek Lang, Francis Donaldson, Malcolm J. LEWIS and Mary J. Wilson. Their work appears in journals such as Biochemical Pharmacology, Advances in Therapy, The Journal of Immunology, Respiration and International Journal of COPD.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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