James E. Hayden
Impact in
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- Cancer, Hypoxia, and Metabolism
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- Proteoglycans and glycosaminoglycans research
Papers in
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- Genomics and Chromatin Dynamics 4
- RNA Research and Splicing 3
- DNA Repair Mechanisms 1
- Epigenetics and DNA Methylation 1
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- Microtubule and mitosis dynamics 2
- Co-authors
- Paul Mrass (1 shared paper)Michele H. Jacob (1 shared paper)Sonali Majumdar (1 shared paper)Wolfgang Weninger (1 shared paper)Richard K. Assoian (1 shared paper)Ellen Puré (1 shared paper)Paul M. Lieberman (5 shared papers)Andrew V. Kossenkov (3 shared papers)
- Journals
- Journal of Cell Science (3 papers)PLoS Pathogens (3 papers)Nature Communications (2 papers)PLoS ONE (1 paper)Molecular Biology of the Cell (1 paper)
- Partner nations
- United StatesGermanyItaly
In The Last Decade
James E. Hayden
11 papers receiving 428 citations
Peers
Comparison fields: 5 of 68
- Cancer Research 64
- Cell Biology 72
- Molecular Biology 260
- Oncology 87
- Immunology and Allergy 19
Countries citing papers authored by James E. Hayden
This map shows the geographic impact of James E. Hayden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Hayden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Hayden more than expected).
Fields of papers citing papers by James E. Hayden
This network shows the impact of papers produced by James E. Hayden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Hayden. The network helps show where James E. Hayden may publish in the future.
Co-authors
The 25 scholars most cited alongside James E. Hayden, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2008 | 138 | |
| 2 | 2016 | 107 | |
| 3 | 2023 | 38 | |
| 4 | 2021 | 30 | |
| 5 | 2019 | 29 | |
| 6 | 2016 | 23 | |
| 7 | 2011 | 22 | |
| 8 | 2019 | 20 | |
| 9 | 2013 | 17 | |
| 10 | 2022 | 9 | |
| 11 | 2025 | 6 | |
| 12 | 2025 | 0 |
About James E. Hayden
James E. Hayden is a scholar working on Molecular Biology, Cell Biology, Oncology, Epidemiology and Cancer Research, having authored 12 papers that have together received 439 indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (4 papers), Viral-associated cancers and disorders (3 papers), RNA Research and Splicing (3 papers), Cytomegalovirus and herpesvirus research (3 papers), Microtubule and mitosis dynamics (2 papers), Cancer-related molecular mechanisms research (1 paper), DNA Repair Mechanisms (1 paper) and Epigenetics and DNA Methylation (1 paper). The work is most often cited by research in Cancer Research (64 citations), Cell Biology (72 citations), Molecular Biology (260 citations), Oncology (87 citations) and Immunology and Allergy (19 citations). James E. Hayden has collaborated with scholars based in United States, Germany and Italy. Frequent co-authors include Paul Mrass, Michele H. Jacob, Sonali Majumdar, Wolfgang Weninger, Richard K. Assoian, Ellen Puré, Paul M. Lieberman, Andrew V. Kossenkov, Andrew R. Cohen and Zhong Deng. Their work appears in journals such as Journal of Cell Science, PLoS Pathogens, Nature Communications, PLoS ONE and Molecular Biology of the Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.