James E. Hayden
Impact in
- Oncology top 5%
- Cancer Immunotherapy and Biomarkers
- Cancer Cells and Metastasis
- Cutaneous Melanoma Detection and Management
- Molecular Biology top 5%
- Melanoma and MAPK Pathways
- Mitochondrial Function and Pathology
- Protein Kinase Regulation and GTPase Signaling
Papers in
-
- Proteoglycans and glycosaminoglycans research 2
- Microtubule and mitosis dynamics 1
- Oncology 5
- Viral-associated cancers and disorders 3
- Cancer Immunotherapy and Biomarkers 1
- Co-authors
- Wolfgang Weninger (1 shared paper)Richard K. Assoian (1 shared paper)Sonali Majumdar (1 shared paper)Ellen Puré (1 shared paper)Paul Mrass (1 shared paper)Paul M. Lieberman (5 shared papers)Andrew V. Kossenkov (3 shared papers)Zhong Deng (3 shared papers)
- Journals
- Nature Communications (3 papers)PLoS Pathogens (3 papers)Journal of Cell Science (3 papers)Molecular Biology of the Cell (1 paper)PLoS ONE (1 paper)
- Partner nations
- United StatesGermanyItaly
In The Last Decade
James E. Hayden
14 papers receiving 2k citations
James E. Hayden's Hit Papers
Peers
Comparison fields: 5 of 75
- Oncology 431
- Molecular Biology 1.2k
- Cell Biology 160
- Cancer Research 141
- Aging 13
Countries citing papers authored by James E. Hayden
This map shows the geographic impact of James E. Hayden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Hayden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Hayden more than expected).
Fields of papers citing papers by James E. Hayden
This network shows the impact of papers produced by James E. Hayden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Hayden. The network helps show where James E. Hayden may publish in the future.
Co-authors
The 25 scholars most cited alongside James E. Hayden, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Acquired Resistance to BRAF Inhibitors Mediated by a RAF Kinase Switch in Melanoma Can Be Overcome by Cotargeting MEK and IGF-1R/PI3K Hit paper breakdown → | 2010 | 1071 |
| 2 | Remodeling of the Collagen Matrix in Aging Skin Promotes Melanoma Metastasis and Affects Immune Cell Motility Hit paper breakdown → | 2018 | 356 |
| 3 | 2021 | 154 | |
| 4 | 2008 | 139 | |
| 5 | 2016 | 118 | |
| 6 | 2023 | 43 | |
| 7 | 2021 | 35 | |
| 8 | 2019 | 31 | |
| 9 | 2011 | 22 | |
| 10 | 2016 | 22 | |
| 11 | 2019 | 22 | |
| 12 | 2013 | 17 | |
| 13 | 2022 | 12 | |
| 14 | 2025 | 6 | |
| 15 | 2025 | 0 |
About James E. Hayden
James E. Hayden is a scholar working on Cell Biology, Oncology, Molecular Biology, Urology and Dermatology, having authored 15 papers that have together received 2k indexed citations. Recurring topics across this work include Genomics and Chromatin Dynamics (4 papers), Viral-associated cancers and disorders (3 papers), TGF-β signaling in diseases (2 papers), Proteoglycans and glycosaminoglycans research (2 papers), Cancer-related molecular mechanisms research (1 paper), Melanoma and MAPK Pathways (1 paper), Microtubule and mitosis dynamics (1 paper) and Cancer Immunotherapy and Biomarkers (1 paper). The work is most often cited by research in Oncology (431 citations), Molecular Biology (1.2k citations), Cell Biology (160 citations), Cancer Research (141 citations) and Aging (13 citations). James E. Hayden has collaborated with scholars based in United States, Germany and Italy. Frequent co-authors include Wolfgang Weninger, Richard K. Assoian, Sonali Majumdar, Ellen Puré, Paul Mrass, Paul M. Lieberman, Andrew V. Kossenkov, Zhong Deng, Andrew R. Cohen and Andreas Wiedmer. Their work appears in journals such as Nature Communications, PLoS Pathogens, Journal of Cell Science, Molecular Biology of the Cell and PLoS ONE.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.