Inès Schmid
Impact in
- Immunology top 2%
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms
- Immune Response and Inflammation
- Immune cells in cancer
- Epidemiology top 2%
- Autophagy in Disease and Therapy
Papers in
-
- RNA Interference and Gene Delivery 2
- Studies on Chitinases and Chitosanases 1
- DNA Repair Mechanisms 1
-
- Immune Response and Inflammation 4
- Neutrophil, Myeloperoxidase and Oxidative Mechanisms 2
- Immune Cell Function and Interaction 2
- Co-authors
- Hans‐Uwe Simon (10 shared papers)Shída Yousefi (9 shared papers)Esther Kozlowski (1 shared paper)Andrew Ziemiecki (2 shared papers)Léonardo Scapozza (2 shared papers)Thomas Schaffner (1 shared paper)Thomas Brunner (1 shared paper)Remo Perozzo (1 shared paper)
- Journals
- The Journal of Immunology (2 papers)Nature Communications (1 paper)Nature Medicine (1 paper)Allergy (1 paper)Molecular Cell (1 paper)
- Partner nations
- SwitzerlandCzechiaBelgium
In The Last Decade
Inès Schmid
12 papers receiving 3.2k citations
Inès Schmid's Hit Papers
Peers
Comparison fields: 5 of 106
- Immunology 1.4k
- Epidemiology 1.1k
- Physiology 128
- Immunology and Allergy 140
- Physiology 443
Countries citing papers authored by Inès Schmid
This map shows the geographic impact of Inès Schmid's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Inès Schmid with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Inès Schmid more than expected).
Fields of papers citing papers by Inès Schmid
This network shows the impact of papers produced by Inès Schmid. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Inès Schmid. The network helps show where Inès Schmid may publish in the future.
Co-authors
The 25 scholars most cited alongside Inès Schmid, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Calpain-mediated cleavage of Atg5 switches autophagy to apoptosis Hit paper breakdown → | 2006 | 1078 |
| 2 | Viable neutrophils release mitochondrial DNA to form neutrophil extracellular traps Hit paper breakdown → | 2009 | 784 |
| 3 | Catapult-like release of mitochondrial DNA by eosinophils contributes to antibacterial defense Hit paper breakdown → | 2008 | 741 |
| 4 | 2005 | 169 | |
| 5 | 2004 | 155 | |
| 6 | 2013 | 122 | |
| 7 | 2015 | 88 | |
| 8 | 2012 | 20 | |
| 9 | 2009 | 16 | |
| 10 | 2009 | 15 | |
| 11 | 2002 | 5 | |
| 12 | 2008 | 1 |
About Inès Schmid
Inès Schmid is a scholar working on Molecular Biology, Immunology, Epidemiology, Rheumatology and Cell Biology, having authored 12 papers that have together received 3.2k indexed citations. Recurring topics across this work include Immune Response and Inflammation (4 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), RNA Interference and Gene Delivery (2 papers), Autophagy in Disease and Therapy (2 papers), Immune Cell Function and Interaction (2 papers), Eosinophilic Disorders and Syndromes (2 papers), Studies on Chitinases and Chitosanases (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Immunology (1.4k citations), Epidemiology (1.1k citations), Physiology (128 citations), Immunology and Allergy (140 citations) and Physiology (443 citations). Inès Schmid has collaborated with scholars based in Switzerland, Czechia and Belgium. Frequent co-authors include Hans‐Uwe Simon, Shída Yousefi, Esther Kozlowski, Andrew Ziemiecki, Léonardo Scapozza, Thomas Schaffner, Thomas Brunner, Remo Perozzo, Alex Straumann and Nicola Andina. Their work appears in journals such as The Journal of Immunology, Nature Communications, Nature Medicine, Allergy and Molecular Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.