Ilker Karaca
Impact in
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- Neuroinflammation and Neurodegeneration Mechanisms
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- Alzheimer's disease research and treatments
- Lysosomal Storage Disorders Research
Papers in
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- Alzheimer's disease research and treatments 4
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- Neuroinflammation and Neurodegeneration Mechanisms 3
- Parkinson's Disease Mechanisms and Treatments 1
- Co-authors
- Jochen Walter (7 shared papers)Irfan Y. Tamboli (2 shared papers)Gerhild van Echten‐Deckert (2 shared papers)Nguyen T. Tien (1 shared paper)Konstantin Glebov (2 shared papers)Patrick Wunderlich (1 shared paper)Tobias Hartmann (1 shared paper)Marcus O.W. Grimm (1 shared paper)
- Journals
- Journal of Biological Chemistry (2 papers)Brain Behavior and Immunity (1 paper)Pharmacopsychiatry (1 paper)Journal of Neuroinflammation (1 paper)Cellular Physiology and Biochemistry (1 paper)
- Partner nations
- Germany
In The Last Decade
Ilker Karaca
8 papers receiving 202 citations
Peers
Comparison fields: 5 of 50
- Neurology 49
- Physiology 94
- Cell Biology 44
- Biological Psychiatry 6
- Aging 4
Countries citing papers authored by Ilker Karaca
This map shows the geographic impact of Ilker Karaca's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ilker Karaca with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ilker Karaca more than expected).
Fields of papers citing papers by Ilker Karaca
This network shows the impact of papers produced by Ilker Karaca. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ilker Karaca. The network helps show where Ilker Karaca may publish in the future.
Co-authors
The 25 scholars most cited alongside Ilker Karaca, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2016 | 61 | |
| 2 | 2014 | 55 | |
| 3 | 2014 | 47 | |
| 4 | 2016 | 35 | |
| 5 | 2013 | 3 | |
| 6 | 2017 | 2 | |
| 7 | 2018 | 1 | |
| 8 | 2015 | 1 |
About Ilker Karaca
Ilker Karaca is a scholar working on Physiology, Neurology, Molecular Biology, Oncology and Neurology, having authored 8 papers that have together received 205 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers), Chemokine receptors and signaling (2 papers), Sphingolipid Metabolism and Signaling (2 papers), Parkinson's Disease Mechanisms and Treatments (1 paper), Autophagy in Disease and Therapy (1 paper), GDF15 and Related Biomarkers (1 paper) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in Neurology (49 citations), Physiology (94 citations), Cell Biology (44 citations), Biological Psychiatry (6 citations) and Aging (4 citations). Ilker Karaca has collaborated with scholars based in Germany. Frequent co-authors include Jochen Walter, Irfan Y. Tamboli, Gerhild van Echten‐Deckert, Nguyen T. Tien, Konstantin Glebov, Patrick Wunderlich, Tobias Hartmann, Marcus O.W. Grimm, Viola J. Haupenthal and Markus H. Gräler. Their work appears in journals such as Journal of Biological Chemistry, Brain Behavior and Immunity, Pharmacopsychiatry, Journal of Neuroinflammation and Cellular Physiology and Biochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.