I. Kupfer

411 citations
11 papers · 155 · h-index 6

Impact in

Papers in

I. Kupfer

10 papers receiving 150 citations

Peers

I. Kupfer
Comparison fields: 5 of 33
  • Dermatology 49
  • Pathology and Forensic Medicine 36
  • Immunology 26
  • Hematology 13
  • Neurology 16
Replace L. Méry‐Bossard with:
L. Méry‐Bossard France
Cristina Mordenti Italy
Guillaume Direz France
Elvira Mora Spain
J. Delaleu France
C. Mavragani Greece
Issam Hamadah Saudi Arabia
Janie M. Leonhardt United States
Mathew Rose United States
Michela Tabanelli Italy
I. Kupfer relative to L. Méry‐Bossard France L. Méry‐Bossard's profile →
Citations per field
00.5×3.3×
L. Méry‐Bossard · 1×
Citations per year

Countries citing papers authored by I. Kupfer

Since Specialization
Citations

This map shows the geographic impact of I. Kupfer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I. Kupfer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I. Kupfer more than expected).

Fields of papers citing papers by I. Kupfer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by I. Kupfer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I. Kupfer. The network helps show where I. Kupfer may publish in the future.

Co-authors

The 25 scholars most cited alongside I. Kupfer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with I. Kupfer Line = papers co-authored together I. Kupfer links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 200352
2 201637
3
[Treatment of familial erythermalgia with the association of lidocaine and mexiletine].
200326
4 200419
5 20067
6 20016
7 20025
8 20041
9
[Medium-term renal involvement in adult patients with Henoch-Schönlein purpura].
20001
10 20131
11 20200

About I. Kupfer

I. Kupfer is a scholar working on Dermatology, Pulmonary and Respiratory Medicine, Immunology, Infectious Diseases and Pathology and Forensic Medicine, having authored 11 papers that have together received 155 indexed citations. Recurring topics across this work include Chemotherapy-related skin toxicity (2 papers), Streptococcal Infections and Treatments (1 paper), Autoimmune Neurological Disorders and Treatments (1 paper), Vasculitis and related conditions (1 paper), Genetic and Kidney Cyst Diseases (1 paper), Skin Diseases and Diabetes (1 paper), Mast cells and histamine (1 paper) and Psoriasis: Treatment and Pathogenesis (1 paper). The work is most often cited by research in Dermatology (49 citations), Pathology and Forensic Medicine (36 citations), Immunology (26 citations), Hematology (13 citations) and Neurology (16 citations). I. Kupfer has collaborated with scholars based in France, Belgium and Réunion. Frequent co-authors include P. Joly, Philippe Courville, X. Balguérie, B. Sassolas, L. Misery, G. Guillet, E. Mahé, Alain Beauchet, Alice Phan and Maryam Piram. Their work appears in journals such as British Journal of Dermatology, Pediatric Dermatology, Journal of the American Academy of Dermatology, Annales de Dermatologie et de Vénéréologie and Cancer Genetics and Cytogenetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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