Gerald D. Frank

3.3k citations

43 papers · 2.8k · h-index 33

Impact in

Cardiology and Cardiovascular Medicine top 2%
- Renin-Angiotensin System Studies
Endocrinology, Diabetes and Metabolism top 2%
- Hormonal Regulation and Hypertension

Papers in

Molecular Biology 24
- Protein Kinase Regulation and GTPase Signaling 14
- Receptor Mechanisms and Signaling 6
- PI3K/AKT/mTOR signaling in cancer 5
Cardiology and Cardiovascular Medicine 11
- Renin-Angiotensin System Studies 11

Co-authors: Satoru Eguchi (36 shared papers)Evangeline D. Motley (20 shared papers)Tadashi Inagami (20 shared papers)Haruhiko Ohtsu (12 shared papers)Hiroyuki Suzuki (10 shared papers)Peter J. Dempsey (8 shared papers)Sadaharu Higuchi (9 shared papers)Kunie Eguchi (12 shared papers)
Journals: Hypertension (6 papers)Journal of Biological Chemistry (5 papers)Biochemical and Biophysical Research Communications (4 papers)Arteriosclerosis Thrombosis and Vascular Biology (4 papers)Endocrinology (4 papers)
Partner nations: United States Japan China

In The Last Decade

Gerald D. Frank

43 papers receiving 2.8k citations

Peers

Countries citing papers authored by Gerald D. Frank

Since Specialization

Citations

This map shows the geographic impact of Gerald D. Frank's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gerald D. Frank with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gerald D. Frank more than expected).

Fields of papers citing papers by Gerald D. Frank

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gerald D. Frank. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gerald D. Frank. The network helps show where Gerald D. Frank may publish in the future.

Co-authors

The 25 scholars most cited alongside Gerald D. Frank, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gerald D. Frank Line = papers co-authored together Gerald D. Frank links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

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#	Work
1	Angiotensin II signal transduction through the AT1 receptor: novel insights into mechanisms and pathophysiology Clinical Science ·Sadaharu Higuchi, Haruhiko Ohtsu, Hiroyuki Suzuki, Heigoro Shirai, Gerald D. Frank, Satoru Eguchi	2007	341
2	Activation of MAPKs by Angiotensin II in Vascular Smooth Muscle Cells Journal of Biological Chemistry ·Satoru Eguchi, Peter J. Dempsey, Gerald D. Frank, Evangeline D. Motley, Tadashi Inagami	2001	243
3	Intracellular Signaling of Angiotensin II-induced p70 S6 Kinase Phosphorylation at Ser411 in Vascular Smooth Muscle Cells Journal of Biological Chemistry ·Satoru Eguchi, Hiroaki Iwasaki, Hikaru Ueno, Gerald D. Frank, Evangeline D. Motley, Kunie Eguchi, Fumiaki Marumo, Yukio Hirata, Tadashi Inagami	1999	150
4	Angiotensin II–Induced Cardiac Hypertrophy and Hypertension Are Attenuated by Epidermal Growth Factor Receptor Antisense Circulation ·Shuntaro Kagiyama, Satoru Eguchi, Gerald D. Frank, Tadashi Inagami, Yuan Zhang, M. Ian Phillips	2002	141
5	ADAM17 Mediates Epidermal Growth Factor Receptor Transactivation and Vascular Smooth Muscle Cell Hypertrophy Induced by Angiotensin II Arteriosclerosis Thrombosis and Vascular Biology ·Haruhiko Ohtsu, Peter J. Dempsey, Gerald D. Frank, Eugen Brailoiu, Sadaharu Higuchi, Hiroyuki Suzuki, H. Nakashima, Kunie Eguchi, Satoru Eguchi	2006	118
6	Angiotensin II Regulates Vascular and Endothelial Dysfunction: Recent Topics of Angiotensin II Type-1 Receptor Signaling in the Vasculature Current Vascular Pharmacology ·H. Nakashima, Hiroyuki Suzuki, Haruhiko Ohtsu, James Chao, Hirotoshi Utsunomiya, Gerald D. Frank, Satoru Eguchi	2005	111
7	Involvement of Reactive Oxygen Species in the Activation of Tyrosine Kinase and Extracellular Signal-Regulated Kinase by Angiotensin II¹ Endocrinology ·Gerald D. Frank, Satoru Eguchi, Tadashi Yamakawa, S. Tanaka, Tadashi Inagami, Evangeline D. Motley	2000	105
8	G Protein Coupling and Second Messenger Generation Are Indispensable for Metalloprotease-dependent, Heparin-binding Epidermal Growth Factor Shedding through Angiotensin II Type-1 Receptor Journal of Biological Chemistry ·Mizuo Mifune, Haruhiko Ohtsu, Hiroyuki Suzuki, H. Nakashima, Eugen Brailoiu, Nae J. Dun, Gerald D. Frank, Tadashi Inagami, Shigeki Higashiyama, Walter G. Thomas, Andrea D. Eckhart, Peter J. Dempsey, Satoru Eguchi	2005	104
9	Signal-Crosstalk Between Rho/ROCK and c-Jun NH ₂ -Terminal Kinase Mediates Migration of Vascular Smooth Muscle Cells Stimulated by Angiotensin II Arteriosclerosis Thrombosis and Vascular Biology ·Haruhiko Ohtsu, Mizuo Mifune, Gerald D. Frank, Shuichi Saito, Tadashi Inagami, Shokei Kim‐Mitsuyama, Yoh Takuwa, Terukatsu Sasaki, Jeffrey D. Rothstein, Hiroyuki Suzuki, H. Nakashima, Elethia A. Woolfolk, Evangeline D. Motley, Satoru Eguchi	2005	102
10	Distinct Mechanisms of Receptor and Nonreceptor Tyrosine Kinase Activation by Reactive Oxygen Species in Vascular Smooth Muscle Cells: Role of Metalloprotease and Protein Kinase C-δ Molecular and Cellular Biology ·Gerald D. Frank, Mizuo Mifune, Tadashi Inagami, Motoi Ohba, Terukatsu Sasaki, Shigeki Higashiyama, Peter J. Dempsey, Satoru Eguchi	2003	87
11	Ligand-independent trans-Activation of the Platelet-derived Growth Factor Receptor by Reactive Oxygen Species Requires Protein Kinase C-δ and c-Src Journal of Biological Chemistry ·Shuichi Saito, Gerald D. Frank, Mizuo Mifune, Motoi Ohba, Hirotoshi Utsunomiya, Evangeline D. Motley, Tadashi Inagami, Satoru Eguchi	2002	82
12	Angiotensin II Signal Transduction Through Small GTP-Binding Proteins Hypertension ·Haruhiko Ohtsu, Hiroyuki Suzuki, H. Nakashima, Sudhir Dhobale, Gerald D. Frank, Evangeline D. Motley, Satoru Eguchi	2006	78
13	Requirement of Ca²⁺and PKCδ for Janus Kinase 2 Activation by Angiotensin II: Involvement of PYK2 Molecular Endocrinology ·Gerald D. Frank, Shuichi Saito, Evangeline D. Motley, Terukatsu Sasaki, Motoi Ohba, Toshio Kuroki, Tadashi Inagami, Satoru Eguchi	2002	65
14	Hydrogen Peroxide Inhibits Insulin Signaling in Vascular Smooth Muscle Cells Experimental Biology and Medicine ·Carla D. Gardner, Satoru Eguchi, Cherilynn M. Reynolds, Kunie Eguchi, Gerald D. Frank, Evangeline D. Motley	2003	61
15	Lysophosphatidylcholine Inhibits Insulin-Induced Akt Activation Through Protein Kinase C-α in Vascular Smooth Muscle Cells Hypertension ·Evangeline D. Motley, Syeda M. Kabir, Carla D. Gardner, Kunie Eguchi, Gerald D. Frank, Toshio Kuroki, Motoi Ohba, Tadashi Yamakawa, Satoru Eguchi	2002	58
16	Insulin-Induced Akt Activation Is Inhibited by Angiotensin II in the Vasculature Through Protein Kinase C-α Hypertension ·Evangeline D. Motley, Kunie Eguchi, Carla D. Gardner, Adrienne L. Hicks, Cherilynn M. Reynolds, Gerald D. Frank, Mizuo Mifune, Motoi Ohba, Satoru Eguchi	2003	58
17	Activation of Tyrosine Kinases by Reactive Oxygen Species in Vascular Smooth Muscle Cells: Significance and Involvement of EGF Receptor Transactivation by Angiotensin II Antioxidants and Redox Signaling ·Gerald D. Frank, Satoru Eguchi	2003	58
18	Basic polyamino acids rich in arginine, lysine, or ornithine cause both enhancement of and refractoriness to formation of endothelium-derived nitric oxide in pulmonary artery and vein. Circulation Research ·Louis J. Ignarro, Michele E. Gold, Georgette M. Buga, R E Byrns, K S Wood, Gautam Chaudhuri, Gerald D. Frank	1989	53
19	Metalloprotease inhibitor blocks angiotensin II-induced migration through inhibition of epidermal growth factor receptor transactivation Biochemical and Biophysical Research Communications ·Shuichi Saito, Gerald D. Frank, Evangeline D. Motley, Peter J. Dempsey, Hirotoshi Utsunomiya, Tadashi Inagami, Satoru Eguchi	2002	52
20	PYK2/CAKβ Represents a Redox-Sensitive Tyrosine Kinase in Vascular Smooth Muscle Cells Biochemical and Biophysical Research Communications ·Gerald D. Frank, Evangeline D. Motley, Tadashi Inagami, Satoru Eguchi	2000	50

About Gerald D. Frank

Gerald D. Frank is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Oncology, Physiology and Immunology and Allergy, having authored 43 papers that have together received 2.8k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (14 papers), Renin-Angiotensin System Studies (11 papers), Nitric Oxide and Endothelin Effects (10 papers), Receptor Mechanisms and Signaling (6 papers), Cytokine Signaling Pathways and Interactions (6 papers), PI3K/AKT/mTOR signaling in cancer (5 papers), HER2/EGFR in Cancer Research (4 papers) and Cell Adhesion Molecules Research (4 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (830 citations), Endocrinology, Diabetes and Metabolism (403 citations), Immunology and Allergy (146 citations), Physiology (543 citations) and Molecular Biology (1.4k citations). Gerald D. Frank has collaborated with scholars based in United States, Japan and China. Frequent co-authors include Satoru Eguchi, Evangeline D. Motley, Tadashi Inagami, Haruhiko Ohtsu, Hiroyuki Suzuki, Peter J. Dempsey, Sadaharu Higuchi, Kunie Eguchi, Heigoro Shirai and H. Nakashima. Their work appears in journals such as Hypertension, Journal of Biological Chemistry, Biochemical and Biophysical Research Communications, Arteriosclerosis Thrombosis and Vascular Biology and Endocrinology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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