Gavin E. Greenoak

24 papers receiving 884 citations

Peers

Gavin E. Greenoak
Comparison fields: 5 of 108
  • Dermatology 377
  • Health, Toxicology and Mutagenesis 148
  • Pharmaceutical Science 59
  • Cell Biology 115
  • Urology 39
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Countries citing papers authored by Gavin E. Greenoak

Since Specialization
Citations

This map shows the geographic impact of Gavin E. Greenoak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Gavin E. Greenoak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Gavin E. Greenoak more than expected).

Fields of papers citing papers by Gavin E. Greenoak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Gavin E. Greenoak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Gavin E. Greenoak. The network helps show where Gavin E. Greenoak may publish in the future.

Co-authors

The 25 scholars most cited alongside Gavin E. Greenoak, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Gavin E. Greenoak Line = papers co-authored together Gavin E. Greenoak links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 24 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2010232
2 1988174
3 198973
4 201267
5 198466
6
A comparative study of fabric protection against ultraviolet-induced erythema determined by spectrophotometric and human skin measurements.
199136
7 201535
8 200031
9 198830
10 199623
11 199323
12 198221
13 198916
14 199012
15 198812
16 200411
17 19859
18
Uv radiation induced murine tumors produced in the absence of uv radiation induced systemic tumor immunosuppression
19918
19
Ultraviolet radiation-induced murine tumors produced in the absence of ultraviolet radiation-induced systemic tumor immunosuppression.
19917
20 19867

About Gavin E. Greenoak

Gavin E. Greenoak is a scholar working on Dermatology, Molecular Biology, Oncology, Cell Biology and Health, Toxicology and Mutagenesis, having authored 24 papers that have together received 912 indexed citations. Recurring topics across this work include Skin Protection and Aging (14 papers), melanin and skin pigmentation (5 papers), Cancer and Skin Lesions (3 papers), Indoor Air Quality and Microbial Exposure (2 papers), CRISPR and Genetic Engineering (2 papers), Ocular Oncology and Treatments (2 papers), Nonmelanoma Skin Cancer Studies (2 papers) and Carcinogens and Genotoxicity Assessment (1 paper). The work is most often cited by research in Dermatology (377 citations), Health, Toxicology and Mutagenesis (148 citations), Pharmaceutical Science (59 citations), Cell Biology (115 citations) and Urology (39 citations). Gavin E. Greenoak has collaborated with scholars based in Australia, Iran and Hong Kong. Frequent co-authors include C. H. Gallagher, Vivienne E. Reeve, Chris Moran, Sabine Fenner, Jonathan P. Stoye, John M. Coffin, Maxine J. McCall, Michael J. Korsch, Malcolm T. McCulloch and Brian L. Gulson. Their work appears in journals such as Photochemistry and Photobiology, Pathology, Journal of Investigative Dermatology, The Science of The Total Environment and Journal of Cellular Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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