G E Conner

747 citations
13 papers · 685 · h-index 11

Impact in

Papers in

    • Ubiquitin and proteasome pathways 3
    • Glycosylation and Glycoproteins Research 2
    • Ion channel regulation and function 2
    • Cellular transport and secretion 3

G E Conner

13 papers receiving 626 citations

Peers

G E Conner
Comparison fields: 5 of 77
  • Cell Biology 183
  • Cancer Research 117
  • Physiology 29
  • Molecular Biology 416
  • Physiology 131
Replace Elena Friedmann with:
Elena Friedmann Switzerland
N Takami Japan
E Springer United States
R.P. Creagan United States
Alison J. Darmon Canada
Soyeong Sim United States
Angel Ashikov Germany
Sumathi Muthukkumar United States
Keiko Hata Japan
Lutz Heine Germany
G E Conner relative to Elena Friedmann Switzerland Elena Friedmann's profile →
Citations per field
00.5×1.5×1.9×
Elena Friedmann · 1×
Citations per year

Countries citing papers authored by G E Conner

Since Specialization
Citations

This map shows the geographic impact of G E Conner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by G E Conner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites G E Conner more than expected).

Fields of papers citing papers by G E Conner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by G E Conner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by G E Conner. The network helps show where G E Conner may publish in the future.

Co-authors

The 21 scholars most cited alongside G E Conner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with G E Conner Line = papers co-authored together G E Conner links everyone, so they are left out of the graph.

All Works

13 of 13 papers shown
#Work
1 1981175
2 199490
3 199486
4 198269
5 198960
6 198355
7 199236
8 198430
9 198929
10 201427
11 199517
12 19928
13 19903

About G E Conner

G E Conner is a scholar working on Molecular Biology, Cell Biology, Cellular and Molecular Neuroscience, Cardiology and Cardiovascular Medicine and Epidemiology, having authored 13 papers that have together received 685 indexed citations. Recurring topics across this work include Ubiquitin and proteasome pathways (3 papers), Cellular transport and secretion (3 papers), Glycosylation and Glycoproteins Research (2 papers), Ion channel regulation and function (2 papers), Inflammatory Myopathies and Dermatomyositis (2 papers), Viral Infections and Immunology Research (2 papers), Protease and Inhibitor Mechanisms (2 papers) and Systemic Sclerosis and Related Diseases (1 paper). The work is most often cited by research in Cell Biology (183 citations), Cancer Research (117 citations), Physiology (29 citations), Molecular Biology (416 citations) and Physiology (131 citations). G E Conner has collaborated with scholars based in United States. Frequent co-authors include Günter Blobel, Ann H. Erickson, Yi Zhu, Henry G. Kunkel, David E. Fisher, Westley H. Reeves, Matthias Salathé, D. S. Nelson, Rosanne Wisniewolski and R G Lahita. Their work appears in journals such as Journal of Biological Chemistry, Proceedings of the National Academy of Sciences, American Journal of Respiratory and Critical Care Medicine, Biochemical Journal and Neuropeptides.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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