Fei Sa

438 citations
9 papers · 377 · h-index 8

Impact in

Papers in

    • Plant-derived Lignans Synthesis and Bioactivity 2
    • Signaling Pathways in Disease 1
    • Parkinson's Disease Mechanisms and Treatments 4
    • Neuroinflammation and Neurodegeneration Mechanisms 1

Fei Sa

9 papers receiving 371 citations

Peers

Fei Sa
Comparison fields: 5 of 70
  • Complementary and alternative medicine 59
  • Neurology 58
  • Biological Psychiatry 14
  • Neurology 63
  • Cellular and Molecular Neuroscience 61
Replace Hefeng Zhou with:
Hefeng Zhou China
Jarbas Mota Siqueira Brazil
Samuel S.W. Szeto Canada
Hoon Jae Jeong South Korea
Rui Tan China
Hideshi Kaneko Japan
Jia Fan China
Mauro Nicolau Brazil
Manisha Mishra Singapore
Yuanchu Liu China
Fei Sa relative to Hefeng Zhou China Hefeng Zhou's profile →
Citations per field
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Citations per year

Countries citing papers authored by Fei Sa

Since Specialization
Citations

This map shows the geographic impact of Fei Sa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fei Sa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fei Sa more than expected).

Fields of papers citing papers by Fei Sa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fei Sa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fei Sa. The network helps show where Fei Sa may publish in the future.

Co-authors

The 25 scholars most cited alongside Fei Sa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Fei Sa Line = papers co-authored together Fei Sa links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 201371
2 200765
3 201561
4 201255
5 201539
6 201139
7 201522
8 201519
9 20136

About Fei Sa

Fei Sa is a scholar working on Molecular Biology, Neurology, Pharmacology, Complementary and alternative medicine and Physiology, having authored 9 papers that have together received 377 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (4 papers), Cholinesterase and Neurodegenerative Diseases (2 papers), Traditional Chinese Medicine Analysis (2 papers), Plant-derived Lignans Synthesis and Bioactivity (2 papers), Cancer Genomics and Diagnostics (1 paper), Alzheimer's disease research and treatments (1 paper), Signaling Pathways in Disease (1 paper) and Neuroinflammation and Neurodegeneration Mechanisms (1 paper). The work is most often cited by research in Complementary and alternative medicine (59 citations), Neurology (58 citations), Biological Psychiatry (14 citations), Neurology (63 citations) and Cellular and Molecular Neuroscience (61 citations). Fei Sa has collaborated with scholars based in Macao, Hong Kong and China. Frequent co-authors include Simon Ming‐Yuen Lee, Maggie Pui Man Hoi, Ying Zheng, Zhong-Yan Zhou, Yitao Wang, Kwok‐Pui Fung, Jian‐Li Gao, Cheong‐Meng Chong, Sai Li and Kelvin Chan. Their work appears in journals such as Parkinson s Disease, Molecular BioSystems, Chemico-Biological Interactions, British Journal of Pharmacology and Free Radical Biology and Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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