Eva Westphal

457 citations
10 papers · 418 · h-index 8

Impact in

Papers in

    • Viral-associated cancers and disorders 4
    • CRISPR and Genetic Engineering 2
    • Ubiquitin and proteasome pathways 1

Eva Westphal

10 papers receiving 407 citations

Peers

Eva Westphal
Comparison fields: 5 of 58
  • Oncology 276
  • Pathology and Forensic Medicine 73
  • Immunology 65
  • Epidemiology 94
  • Transplantation 6
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Citations per year

Countries citing papers authored by Eva Westphal

Since Specialization
Citations

This map shows the geographic impact of Eva Westphal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Westphal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Westphal more than expected).

Fields of papers citing papers by Eva Westphal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Westphal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Westphal. The network helps show where Eva Westphal may publish in the future.

Co-authors

The 25 scholars most cited alongside Eva Westphal, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Eva Westphal Line = papers co-authored together Eva Westphal links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1
Activation of lytic Epstein-Barr virus (EBV) infection by radiation and sodium butyrate in vitro and in vivo: a potential method for treating EBV-positive malignancies.
2000132
2 199879
3
Induction of lytic Epstein-Barr virus (EBV) infection in EBV-associated malignancies using adenovirus vectors in vitro and in vivo.
199969
4 200248
5 199837
6 199829
7 200210
8
Clinical relevance of cytotoxic T-cell precursor (p-CTL) frequencies in allograft recipients.
19908
9
Self-deleting suicide vectors (SDSV): selective killing of p53-deficient cancer cells.
20015
10 19981

About Eva Westphal

Eva Westphal is a scholar working on Oncology, Molecular Biology, Genetics, Infectious Diseases and Epidemiology, having authored 10 papers that have together received 418 indexed citations. Recurring topics across this work include Viral-associated cancers and disorders (4 papers), Virus-based gene therapy research (4 papers), Parvovirus B19 Infection Studies (3 papers), Cancer Research and Treatments (2 papers), CRISPR and Genetic Engineering (2 papers), Cytomegalovirus and herpesvirus research (2 papers), Chronic Myeloid Leukemia Treatments (1 paper) and Ubiquitin and proteasome pathways (1 paper). The work is most often cited by research in Oncology (276 citations), Pathology and Forensic Medicine (73 citations), Immunology (65 citations), Epidemiology (94 citations) and Transplantation (6 citations). Eva Westphal has collaborated with scholars based in United States, Germany and Russia. Frequent co-authors include Shannon C. Kenney, William Blackstock, Wenhai Feng, Bruce F. Israel, Amy Mauser, Christine L. Talarico, M G Davis, Jessica Swenson, Kuang‐Ming Hsiao and Yue Xiong. Their work appears in journals such as Human Gene Therapy, Journal of Virology, Molecular and Cellular Biology, Journal of Dermatological Science and Current Pharmaceutical Design.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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