Diane M. Studzinski

467 citations
30 papers · 384 · h-index 12

Impact in

Papers in

Diane M. Studzinski

26 papers receiving 380 citations

Peers

Diane M. Studzinski
Comparison fields: 5 of 60
  • Developmental Neuroscience 102
  • Neurology 86
  • Biological Psychiatry 11
  • Cellular and Molecular Neuroscience 73
  • Molecular Biology 155
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Achilles K. Papavasiliou United States
Daniel Chesik Netherlands
Emily Ren United States
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Citations per year

Countries citing papers authored by Diane M. Studzinski

Since Specialization
Citations

This map shows the geographic impact of Diane M. Studzinski's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Diane M. Studzinski with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Diane M. Studzinski more than expected).

Fields of papers citing papers by Diane M. Studzinski

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Diane M. Studzinski. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Diane M. Studzinski. The network helps show where Diane M. Studzinski may publish in the future.

Co-authors

The 25 scholars most cited alongside Diane M. Studzinski, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Diane M. Studzinski Line = papers co-authored together Diane M. Studzinski links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 30 papers — load more, or switch the sort, to bring in the rest.

#Work
1 199058
2 200936
3 200735
4 198735
5 201135
6 202024
7 199817
8 202117
9 199416
10 199915
11 198614
12 200113
13 198910
14 20219
15 20237
16 20216
17 20096
18 20215
19 20145
20 20124

About Diane M. Studzinski

Diane M. Studzinski is a scholar working on Developmental Neuroscience, Neurology, Pulmonary and Respiratory Medicine, Cellular and Molecular Neuroscience and Surgery, having authored 30 papers that have together received 384 indexed citations. Recurring topics across this work include Neurogenesis and neuroplasticity mechanisms (6 papers), Neuroinflammation and Neurodegeneration Mechanisms (4 papers), Aortic aneurysm repair treatments (3 papers), RNA Research and Splicing (3 papers), Cytokine Signaling Pathways and Interactions (2 papers), RNA regulation and disease (2 papers), Immune Response and Inflammation (2 papers) and MicroRNA in disease regulation (2 papers). The work is most often cited by research in Developmental Neuroscience (102 citations), Neurology (86 citations), Biological Psychiatry (11 citations), Cellular and Molecular Neuroscience (73 citations) and Molecular Biology (155 citations). Diane M. Studzinski has collaborated with scholars based in United States and United Kingdom. Frequent co-authors include Joyce A. Benjamins, Robert P. Skoff, Rose E. Callahan, Liljana Nedelkoska, Beverly Bealmear, Robert P. Lisak, Pamela E. Knapp, Bin Wei Yao, Graham W. Long and Susan J. Land. Their work appears in journals such as Journal of Vascular Surgery, Journal of Neuroinflammation, Journal of Neuroimmunology, The American Journal of Surgery and Journal of Neurochemistry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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